Nawa ne bincike game da alamomin nau'in ciwon sukari na 2?

Duk yana dogara da takamaiman cutar. Farashi ya tashi daga dubu biyu zuwa dubun dubun rubles. Anan gajeriyar jerin cututtukan da aka gwada a UnionMed:

- insulin da nau'in II.

Kudin bincike na DNA zai kuma dogara da yawan cututtukan da aka yi nazari da kuma daki-daki na nazarin abubuwan.

Gwajin kwayoyin yana ba ka damar gano yanayin tsinkaye ga waɗannan cututtukan:

• Hauhawar jini,
• Hawan jini
• Atherosclerosis na aorta da na jijiyoyin jini,
• Hadarin mutuwa kwatsam
• Thrombophilia da varicose veins.
• Binciken duk alamomin katin.

• Type I ciwon sukari mellitus (insulin-dogara),
• Nau'in ciwon siga na II
• Ciwon sukari mai fama da cutar siga a cikin nau'in na ciwon suga.

Cututtuka na numfashi:

• Asma,
• Ciwon mara mai fama da cutar sankara.

Cututtuka na ciki

• Cutar ta Crohn
• Nonspecific ulcerative colitis.

Cututtuka na metabolism kashi:

Cutar kansa ta kansa

• Ciwon kansar
• Kabarin cuta
• Abun ciwon kansa na autoimmune.

• Endometriosis
• Rashin haihuwa
• Lahani na bututun ƙarfe,
• Tsinkaya zuwa Down syndrome a tayin,
• Easy mai tsabta gestosis,
• Mai tsananin tsabtace gestosis,

Bayyanar cututtuka na cututtukan cututtukan da ke faruwa a cikin yara:

• Huntington Chorea,
• Adrenogenital ciwo (na haihuwar 21-hydroxylase rashi),
• Cystic fibrosis,
• Shawnanabe,
• Duchenne myodystrophy,
• Myotonic dystrophy,
• Martin Bell na Ciwon,
• Rashin ƙwayar atrophy na kashin baya (cutar Werdnig-Hoffmann).

Yin gwaje-gwaje a asibitin ARMED shine zabi da ya dace

• Garantin mu amintacciya ce kuma aka amince da ita don gudanar da duk karatun da muke samarwa marasa lafiya,
• Yana da lasisi mai inganci na likita
• Dakin gwaje-gwaje na sanye da kayan zamani,
• Ana bayar da sakamakon gwajin a cikin dan kankanen lokaci kuma likitoci masu gogewa sun yanke hukunci,
• An tabbatar wa mai haƙuri tabbacin sirri.

A cikin dakin gwaje-gwaje na likita na asibitin "ARMED", kuna da sabis ɗinku sabis na bincike da cikakken binciken da suka zama dole don gano lokaci na wasu cututtuka ko karkacewa:

Zaɓi nau'in bincike da kake so.

• Nazarin
• Alamomin cutar sankarau
• Koma baya

Yanayin shirye-shiryen bincike *

Samfura samfurin lokacin *

Sakamakon bayarwa *

Kyawawa da safe, a kan komai a ciki

7 a.m. - 2 p.m. Sat 7 a.m. - 12 p.m. rana. 8 a.m. - 11 a.m.

A ranar isar da kayayyakin tarihin daga 16: 00-19: 00, banda Asabar da Lahadi

7 a.m. - 2 p.m. Sat 7 a.m. - 12 p.m. rana. 8 a.m. - 11 a.m.

A ranar isar da kayayyakin tarihin daga 16: 00-19: 00, banda Asabar da Lahadi

7 a.m. - 12 p.m. Sat 7 a.m. - 10 a.m.

A ranar isar da kayayyakin tarihin daga 16: 00-19: 00, banda Asabar da Lahadi

7 a.m. - 12 p.m. Sat 7 a.m. - 11 a.m. rana. 8 a.m. - 11 a.m.

Ranar isar da kayan halitta bayan awa 6, banda Asabar da Lahadi

7 a.m. - 12 p.m. Sat 7 a.m. - 11 a.m. rana. 8 a.m. - 11 a.m.

A ranar isar da kayayyakin tarihin daga 16: 00-19: 00, banda Asabar da Lahadi

7 a.m. - 12 p.m. Sat 7 a.m. - 11 a.m. rana. 8 a.m. - 11 a.m.

Ranar isar da kayan halitta bayan awa 6, banda Asabar da Lahadi

Kyawawa da safe, a kan komai a ciki

7 a.m. - 6.30 p.m. Sat 7 a.m. - 1 p.m. rana. 8 a.m. - 11 a.m.

A cikin kwanaki 12 na aiki

Kyawawa da safe, a kan komai a ciki

7 a.m. - 12 p.m. Sat 7 a.m. - 11 a.m.

A cikin kwanaki 14 na aiki

* Bayar da lokaci da yanayin samfuran kayan don bincike da bayar da sakamakon a cikin rassa ta waya +7 (861) 205-02-02.

** A cikin mata masu juna biyu, ana yin wannan binciken har zuwa mako na 28 na ciki.

Kallon Cutar Malaria

Medicalungiyar likitocin zamani ta ba da shawarar yin gwaji don kamuwa da cutar siga a wasu sassan jama'a. Da farko dai, ya wajaba ga mutanen da suka kai shekaru 45 ko fiye. Idan sakamakon ya kasance mara kyau, ana gudanar da bincike a cikin kowace shekara uku.

Marasa lafiya a matakin ƙuruciya ya kamata a bi hanyoyin da:

• kiba
• m gado,
• kabilanci ko kabila mallakar wani rukuni,
• ciwon sukari
• hauhawar jini
• haihuwar da nauyinsu ya wuce kilogiram 4.5,
• babban glycemia a kan komai a ciki.

Don yin gwaje-gwaje da zazzagewa da ke tsakiya, ana bada shawara don sanin matakin glucose da haemoglobin A1c. Wannan haemoglobin ne, inda ake hade da kwayar halittar glucose tare da kwayar haemoglobin.

Glycosylated haemoglobin yayi aiki tare da glucose jini. Yana aiki a matsayin mai nuna alamar matakin metabolism na metabolism na watanni uku kafin bincike. Yawan ƙirar HbA1c ya dogara da girman hauhawar jini. Normalization na matakin a cikin jini yana faruwa makonni 4-5 bayan euglycemia.

Adadin HbA1c an ƙaddara idan ya zama dole don sarrafa metabolism da kuma tabbatar da diyyarsa a cikin masu ciwon sukari waɗanda suka daɗe da rashin lafiya.

Siffofin Ganewa

Don yin bincike da aiwatar da cikakkiyar kulawa game da Pathology, kuna buƙatar bin hanyoyin bincike da yawa.

Da farko dai, waɗannan sune litattafan dakin gwaje-gwaje na gargajiya, watau nazarin glucose ta hanyar samin fitsari da jini, da gwaji don gwajin ketones da gwajin haƙuri.

Bugu da kari, ana gudanar da bincike kan:

1. HbA1c,
2. fructosamine
3. microalbumin,
4. fitsari creatinine
5. bayanin martaba.

Akwai ƙarin bincike na binciken cutar sankara, wanda ke ba da damar sarrafa ci gaban ciwon sukari, wannan ma'anar:

• C peptide
• kwayoyin kariya daga insulin
• rigakafi zuwa tsibirin na Langengars da tyrosine phosphatase,
• glutamic acid decarboxylase kwayoyin,
• ghrelin, raschistina, leptin, adiponectin,
• HLA bugawa.

Don sanin ilimin halittar shekaru da yawa shekarun da suka gabata, likitoci sun ba da shawarar yin bincike kan sukari mai azumi. A cikin 'yan shekarun nan, an gano cewa akwai ingantacciyar ma'amala tsakanin matakan sukari na jini, raunin jijiyoyin jiki da kuma matakin ci gaban su, an same shi ba tare da sukari mai azumi ba, amma tare da matsayin karuwarsa bayan cin abinci. Wannan ake kira postprandial hyperglycemia.

Duk alamomin nau'in 1 na ciwon sukari za'a iya raba su kamar haka:

1. kwayoyin
2. rigakafi
3. na rayuwa.

HLA bugawa

Ciwon sukari mellitus, daidai da dabaru na likitancin zamani, yana da matsanancin farawa, amma dogon latent. Matani shida ne da aka san su da samuwar wannan ilimin. Farkon waɗannan sune matakan tsinkayen gado ko rashin ƙwayoyin halittar da ke da alaƙa da ciwon sukari na 1.

Zai dace a nuna cewa kasancewar maganin antigens na HLA, musamman na aji na biyu: DR 3, DR 4, DQ, yana da mahimmanci. Hadarin cutar Pathology a wannan yanayin yana ƙaruwa sau da yawa. A halin yanzu, yanayin gado don bayyanar wata nau'in cuta ta nau'in farko ana ɗaukarsa azaman haɗuwa da abubuwa da yawa na abubuwan halittar al'ada.

Mafi alamun alamun sanarwa game da cutar ta 1 sune maganin HLA. Tsarin haloptypes na halayyar nau'in 1 na ciwon sukari ana samun su ne a cikin kashi 77 cikin mutanen da ke ɗauke da ciwon sukari. 6: yana da nau'ikan halittu masu kariya.

Kwayoyi masu kare cututtukan ƙwayoyin cuta zuwa Kwayoyin Langerhans Islet

Sakamakon ƙirƙirar ƙwayoyin autoantibodies zuwa sel na tsibirin na Langerhans, ƙarshen yana lalacewa, wanda ke haifar da rikicewar insulin da kuma bayyanar hoto mai faɗi na nau'in 1 ciwon sukari.

Wadannan nau'ikan ana iya tantance su ta hanyar asalinsu ko kuma sun bayyana saboda dalilai daban-daban.

Daga cikin abubuwanda aka saba dasu sune:

• ƙwayoyin cuta
• abubuwa masu guba
• daban-daban damuwa.

Cutar na farkon nau'ikan ta halin da ake ciki a wani mataki na ciwon suga ba tare da alamun cutar ba, tana iya wuce shekaru da yawa. Za'a iya bayyanar da haɗin da ɓoye insulin a wannan lokacin ta hanyar nazarin haƙuri haƙuri.

A magani, shari'ar gano irin waɗannan ƙwayoyin cuta shekaru takwas ko fiye da haka kafin bayyanar hoton asibiti na cutar. Ya kamata a yi amfani da ma'anar waɗannan ƙwayoyin rigakafin azaman farkon gano asali na ciwon sukari na 1.

A cikin mutane masu irin wannan ƙwayoyin rigakafi, aikin islet cell yana raguwa da sauri, wanda aka bayyana ta hanyar take hakkin ruɗamin insulin. Idan lokaci ya lalace gaba ɗaya, to, cutar siyarwar cututtukan cututtukan ƙwayar cuta na wannan nau'in ya faru.

Yawancin nazarin sun nuna cewa waɗannan kwayoyin suna cikin 70% na masu amsawa tare da sabon nau'in ciwon sukari na 1. A cikin rukunin marasa kula da masu ciwon sukari, akwai kawai 0.1-0.5% na lokuta na gano maganin ƙwayar cuta.

Hakanan ana iya samun waɗannan ƙwayoyin cuta a cikin dangi na masu ciwon sukari. Wannan rukunin mutane yana da matukar hango cutar. Yawancin karatu sun nuna cewa dangi masu dauke da ƙwayoyin cuta suna haifar da nau'in ciwon sukari na 1 a tsawon lokaci.

Alamar kowane nau'i na nau'in sukari na sukari na 2 shima ya haɗa da wannan binciken. Masana ilimin kimiyya sun tabbatar da cewa ƙaddara matakin waɗannan ƙwayoyin rigakafi a cikin masu ciwon sukari tare da nau'in cuta na biyu yana taimaka wajan ayyana shi a fili tun kafin hoton hoto ya bayyana, kuma yana sauƙaƙe ƙaddamar da allurar insulin. Don haka, a cikin masu ciwon sukari tare da nau'in rashin lafiya na biyu, yana yiwuwa hango ko hasashen ƙarin ci gaba da dogaro da insulin na hormone.

Kwayoyin rigakafi zuwa insulin ana samun su a kusan 40% na mutanen da ke da nau'in ciwon sukari 1. Akwai ra'ayi game da daidaituwa tsakanin ƙwayoyin cuta zuwa insulin da ƙwayoyin rigakafi zuwa sel sel.

Tsohon na iya kasancewa a cikin matakin kamuwa da ciwon sukari kuma tare da farkon alamun alamun cutar 1.

Glutamic acid decarboxylase

Kwanan nan, masana kimiyya sun gano babban maganin, wanda shine manufa don autoantibodies waɗanda ke da alaƙa da ƙirƙirar nau'in ciwon sukari da ke dogara da ciwon sukari. Yana da decarboxylase na glutamic acid.

Wannan acid shine enzyme membrane wanda biosynthesizes neurotransmitter na tsakiyar juyayi tsarin - gamma-aminobutyric acid. An fara gano enzyme a cikin mutanen da ke da matsala na tsarin juyayi.

Magungunan rigakafi zuwa GAD sune alamar bada labari game da gano yanayin cutar sankarau. Saboda haka, babban haɗarin kamuwa da ciwon sukari na 1 ana iya gano shi. Tare da asymptomatic samuwar wannan cutar, ana iya gano kwayoyin cuta zuwa GAD a cikin mutane shekaru bakwai kafin bayyananniyar alamun cutar.

Mafi amintacce kuma mai ba da labari tsakanin masana kimiyya ana ganin shine nazarin lokaci ɗaya na alamomi da yawa a cikin jini. Alamar 1 tana wakiltar 20% na bayanin, alamomi biyu sun nuna 44% na bayanan, kuma alamomi uku suna wakiltar 95% na bayanin.

Alamar Ciwon Kai ta Autoimmune

A cikin masu ciwon sukari, bayanin martaba na autoantibodies ya dogara da jinsi da shekaru. Magungunan rigakafi zuwa rigakafi da rigakafi zuwa sel islet, a matsayin mai mulkin, suna cikin yara sama da na manya. Kwayoyi masu kare cututtukan jini zuwa glutamic acid decarboxylase, a mafi yawan lokuta, ana samun su a cikin mata.

Abubuwan da ake tsammani game da samuwar wasu nau'ikan autoantibodies ana iya tantance su ta hanyar nau'ikan tsarin HLA, saboda autoantibodies zuwa insulin, sel islet da islet antigen 2 galibi ana samunsu a cikin mutane da ke da HLA - DR 4 / DQ 8 (DQA 1 * 0301 / DQB 1) * 0302). A lokaci guda, ƙwayoyin rigakafi don rage ƙwayar cuta ta glutamic acid decarboxylase suna cikin mutane waɗanda ke da ƙwayoyin halittar HLA - DR 3 DQ 2 (DQA 1 * 0501 / DQB 1 * 0201).

Yawancin nau'ikan autoantibodies suna yawanci kasancewa a cikin matasa masu ciwon sukari, yayin da mutanen da ke fama da cututtukan sukari na latent suna da nau'in autoantibody guda ɗaya kawai.

Magungunan rigakafi zuwa glutamic acid decarboxylase suna cikin masu ciwon suga da ke ɗauke da nau'in cutar ta farko, amma kuma yawan tasirin yana da yawa a cikin mutane masu kamuwa da cutar ta biyu.

Determinationudurin waɗannan ƙwayoyin cuta ya sa ya yiwu a gano yawancin lambobin da ke da haɗari, idan wannan shine kawai alamar alama ga yawan balagaggu.

Kudin bincike

Mutanen da ke dauke da cutar sukari suna son sha'awar yawan bincike game da alamomin masu ciwon sukari. Akwai wasu bayanan martaba waɗanda aka bayyana ta hanyar yawancin bincike.

Babban gwajin da ake kira iko da ciwon sukari ya hada da gwajin jini da creatinine.

Bugu da kari, bayanin martaba ya kunshi:

1. bincike mai amfani da haemoglobin,
2. triglycerides
3. jimlar cholesterol
4. HDL cholesterol,
5. LDL cholesterol,
6. urinary albumin
7. alakar,
8. Reberg gwaji,
9. glucose a cikin fitsari.

Kudin irin wannan cikakken bincike kusan 5 dubu rubles ne.

Kallon nunawa ya hada da:

1. nazarin glucose na jini
2. glycated haemoglobin.

Farashin bincike shine kusan 900 rubles.

• kwayoyin kariya daga insulin
• maganin rigakafin ƙwayoyin cuta daga ƙwayoyin cutar tyrosine.
• Glutamate decarboxylase kwayoyin,
• maganin rigakafin ƙwayoyin cuta daga ƙwayoyin cutar tyrosine.

Irin wannan bincike zai cinye har 4 dubu rubles.

Gwajin insulin zai kashe kimanin 450 rubles, gwajin C-peptide zai kashe kusan 350 rubles.

Bayyanar cututtuka yayin daukar ciki

Ana ɗaukar gwajin glucose na jini a kan komai a ciki. Tsoron zai haifar da mai nuna alamar 4.8 mmol / daga yatsa da 5.3 - 6.9 mmol / l daga jijiya. Kafin yin gwaje-gwaje, bai kamata mace ta ci abinci na kimanin awanni 10 ba.

Lokacin ɗaukar tayi, za a iya yin gwajin haƙuri haƙuri. A saboda wannan, mace ta sha g 75 na glcose a cikin gilashin ruwa. Bayan awa 2, ana sake yin gwajin jini. Kafin bincike, ba kwa buƙatar iyakance kanka cikin abinci mai gina jiki. Abincin ya kamata ya saba.

Idan an samo alamun cutar sankara, to bai kamata ku jinkirta tattaunawa da likitan ku ba. Gano cutar a wani matakin farko na taimakawa ci gaba da cutar da ci gaban rikice-rikicen rayuwa. Sakamakon bincike dole ne ya kasance daidai, don wannan kuna buƙatar bin duk ƙa'idodin shirye-shirye don bincike.

Yaya bayyanar cutar ciwon sukari ta gaya wa gwani a cikin bidiyo a wannan labarin.