Kusan aboki na har abada na atherosclerosis a cikin ciwon sukari: yadda za a guji bayyanar mara kyau

Tare da matakan glucose da cholesterol a cikin jini, atherosclerosis masu ciwon sukari na ƙananan ƙarshen yawanci yakan faru, amma filayen atherosclerotic na iya haɓaka a cikin wasu gabobin, irin su zuciya da kwakwalwa. Wannan yana haifar da rikice-rikice na rayuwa ga mai haƙuri, wanda zai iya kawo karshen mummunan abu. Haɗin kai tsakanin atherosclerosis da ciwon sukari cuta ce mai lahani ga tasoshin, wanda ke haifar da saurin ketarewar jini a cikinsu.

Dangane da rubutaccen bayani akan “Cike da cutar atherosclerosis na tasoshin da ke kasan bakin ciki” wanda G. V. Dzyak da E. A. Koval, ciwon sukari (mellitus) ke bayarwa yana taimakawa farkon fara rikitarwar cutar atherosclerosis tare da haɓakar yiwuwar lalatawar jini ko bugun jini bayan shekaru 3 na matakan hawan jini.

Dangantakar waɗannan cututtuka

Tare da ciwon sukari, ganuwar jijiyoyin jiki a cikin jiki duka suna zama daɗaɗɗa kuma suna yin gwaje-gwajen ƙwaƙwalwa akai-akai. Wannan yana tsoratar da adon lipoproteins a saman jikinsu, wanda ya tara kuma daga karshe ya haifar da tarkace na atherosclerotic. Kazalika arteriosclerosis na arteries wanda ya haifar da rikicewar rayuwa saboda yawan sukari a cikin jini da kuma tarin LDL. Rashin rikicewar ƙwayar cuta na capillaries ana haifar da su ta hanyar cututtukan cututtukan cututtukan zuciya tare da cutar raunuka na jijiyoyin jiki na atherosclerotic. Wannan yana haifar da mafi saurin bayyanar rikice rikicewar isasshen zagayawa cikin jini ta hanyar bugun jini ko bugun zuciya na gabobin jiki.

Menene abubuwan da ke haifar da ci gaban atherosclerosis a hade tare da ciwon sukari?

Samuwar ƙwayoyin atherosclerotic plates a cikin ciwon sukari mellitus na faruwa ne sakamakon tasirin jikin ɗan adam na waɗannan abubuwan:

Rashin aiki da ƙarancin abinci mai gina jiki suna haifar da haɓaka filayen jirgi a tasoshin.

• mai narkewa mai narkewa, wanda ke haifar da rashin insulin,
• foci na microtraumatization na jini sakamakon da ciwon sukari angiopathy da mai rauni trophism,
• kiba
• salon rayuwa mara aiki, wanda saboda tsananin cutar sankara ce.

Symptomatology

Bayyanar bayyanar cututtukan atherosclerosis a cikin ciwon sukari na iya bambanta, dangane da wurin da keɓaɓɓe da kuma matsayin toshewar gado na jijiyoyin bugun gini. Idan ƙananan hanjin ya shafa, to mai haƙuri yana jin nauyi a cikin kafafu bayan ɗan gajeren tafiya, yana juyawa cikin lameness, ƙyallen fata da ƙyallen fata, da paresthesias daban-daban a cikin rarrafe da siyarwa. Tare da haɗuwa da nau'in 2 na ciwon sukari da atherosclerosis, alamun rashin isasshen wurare dabam dabam yana ƙaruwa da sauri, kuma marasa lafiya kusan babu ciwo. Wannan shi ne saboda farkon necrosis na jijiya karshen a cikin gabobin saboda yawan jini sukari. Idan filayen atherosclerotic na tasoshin kwakwalwa, lalacewar aikinsu, asarar ƙwaƙwalwar ajiya da ƙwarewar fahimi na iya tasowa, paresis, inna, da rikicewar hankali na iya faruwa. A cikin mawuyacin hali, bugun jini na faruwa tare da mutuwar kwakwalwa.

Idan marasa lafiya suna da yawan sukari, to, cututtukan zuciya na zuciya za su zama asymptomatic, ba tare da haifar da ciwo da adadi ba. Wannan ya faru ne saboda farkon necrosis na neurons sakamakon cututtukan cututtukan zuciya na neuropathy. Hakanan cutar ta kasance sanadiyyar farkon haɓakar infarction myocardial tare da rauni mai aiki na tsarin jijiyoyin jini ko mutuwa.

Matakan bincike

Ana iya zargin atherosclerosis na cutar sankara yayin kasancewar alamomin halayyar marasa lafiya. Don tabbatar da wannan cutar, ana yin gwajin jini don sanin matakin cholesterol, da kuma gabobinsa. Bugu da kari, wajibi ne a yi nazarin matakin sukari a cikin komai a ciki kuma bayan cin abinci. Wannan zai tantance ƙarin abun ciki na glucose a cikin jini da gano ciwon sukari. Angiography zai taimaka wajen gano cin zarafin zubar jini na jijiyoyin jiki, kuma yanayin bangon jirgin zai sa ya yiwu a kimanta duban dan tayi. A matsayin ƙarin hanya, ana yin hoton magnetic resonance tare da gabatarwar farkon sabanin matsakaici, wanda zai gano kunkuntar gado na jijiyoyin bugun gini.

Jiyya na Pathology

Kulawa da masu ciwon sikari na mahaifa an yi niyya ne bisa daidaituwa tsakanin sukarin jini da matakan cholesterol. A saboda wannan, ya zama dole a kawar da manyan abubuwanda ke haifar da keta halayen wadannan alamomin. Hakanan yana da mahimmanci a bi abincin da ke hana mai kitse, abinci mai narkewa, da sauƙin narkewa mai narkewa, wanda ke haifar da ciwon sukari na 2. Idan ya cancanta, yin amfani da allurar insulin don daidaita matsayin glucose, an nuna gumaka da fibrates, wanda ke ba da damar rage yawan ƙwayoyin lipoproteins masu cutarwa a cikin jini da hana haɓakar ƙwayar cutar atherosclerotic.

Kiba da warkarwa na yanayin hauhawar jini, wanda kuma zai iya haifar da lalacewar jijiyoyin jiki da kuma samar da adon cholesterol a kansu. Game da rashin daidaito na matakan Conservative, ana aiwatar da maganin atherosclerosis ta amfani da tsoma bakin tiyata tare da kirkirar kwararar jini na wucin gadi saboda samuwar shunt ko jigon wuri, wanda zai fadada jijiyoyin bugun zuciya a wurin da cutar atherosclerotic plaque.

Atherosclerosis da ciwon sukari suna faruwa ne a cikin mazan.

Yadda za a yi gargaɗi?

Yin rigakafin atherosclerosis da ciwon sukari mellitus shine kulawa da kullun akan cholesterol da sukari na jini. Hakanan yana da mahimmanci don kula da ƙoshin lafiya da jagorantar rayuwa mai amfani. Wajibi ne a kawar da masu shaye-shaye, a ci yadda yakamata kuma cikin tsari. Wajibi ne a ware daga abincin da ake amfani da shi a cikin abinci mai gina jiki, abinci mai dadi, da kuma kayan jikin mutum.

Dangantakar shafewa atherosclerosis da ciwon suga

Kasancewar ciwon sukari yana haifar da yaduwar raunuka na ƙwaƙwalwar ƙwaƙwalwa, myocardium, kodan da tasoshin jijiyoyin ƙananan ƙananan gefen. Wannan yana nuna kanta a cikin nau'in bugun jini, bugun zuciya, hauhawar jini na koda, da kuma faruwar irin wannan mummunan rikice-rikice kamar ƙafar mai ciwon sukari. Sakamakonta shine gangrene, kuma yana faruwa a cikin masu ciwon sukari sau 20 sau da yawa fiye da sauran mutanen.

Hanyar atherosclerosis a cikin ciwon sukari mellitus yana da fasalin halayyar:

• ya bayyana kansa shekaru 10 a baya,
• Saide tare da rikitarwa
• yadawo da sauri
• kusan lokaci guda, cututtukan zuciya, jijiyoyin jini, jijiyoyin mahaifa da jijiyoyin gabobin ciki suna shafi.

Kuma anan shine ƙarin game da lalata atherosclerosis na ƙananan ƙarshen.

Sakamakon ciwon sukari da atherosclerosis a kan bango na jijiyoyin jiki

Tare da ciwon sukari da atherosclerosis, akwai rikice-rikice na yau da kullun - lalata halayen arteries na matsakaici da babba. Ciwon sukari angiopathy yawanci yakan faru tare da tsawan lokaci na cutar, wanda ke tattare da yawan saukad da sukari na jini. A lokaci guda, cutar ta ƙunshi manyan (macroangiopathy) da ƙananan hanyoyin jini (microangiopathy), tare suna kai ga tsarin jijiyoyin jini.

Macroangiopathy yana bayyana ta hanyar jijiyoyin zuciya atherosclerosis, cerebral da na gefe, kuma microangiopathy ya haɗa da canje-canje a cikin retina, parenchyma na kodan da jijiyoyin jini na ƙananan ƙarshen. Bugu da ƙari, babban matakin glucose yana lalata ƙwayoyin jijiya, sabili da haka, tare da lalacewar ƙananan ƙarshen, ana kuma lura da cutar neuropathy.

Kwayar halittar jini a cikin jini tana rusa rufin ciki, yana sauƙaƙa shigar azzakari cikin ƙwayar cuta zuwa ciki da samuwar ƙwayar cholesterol. Bayan haka, sai a cika shi da sinadarin alli, rauni da gutsuttsura. A wannan gaba ne ƙwanƙwasa jini yakan toshe sel daga cikin tasoshin, kuma sassan jikinsu suna ɗaukar jini zuwa ƙananan rassa, suna rufe su.

Me yasa ilimin haɓaka tare da sukari mai yawa

Takamaiman dalilai na cututtukan jijiyoyin jiki a atherosclerosis da ciwon sukari sune:

• Sakamakon karancin insulin akan metabolism mai yawa - kara yawan kwayoyi da gabobin atherogenic, a hankali rage fatima a hanta,
• samuwar masu tsattsauran ra'ayi,
• tsari mai kumburi
• take hakkin mutuncin kwasfa na ciki, raunana kayan aikinta na kariya,
• thrombosis
• na jijiyoyin bugun jini.

Yawan cutar angiopathy kuma yana haifar da kiba, wanda yawancin lokuta ana samun shi a cikin nau'in ciwon sukari na 2, hauhawar jijiyoyi, da hauhawar jini. Halin da ake ciki ya tsananta ta hanyar shan sigari, haɗarin aiki, ƙarancin motsa jiki, a cikin tsofaffi da tsofaffi marasa lafiya, ɗaukar nauyi game da cututtuka biyu.

Me ke faruwa

A cikin ciwon sukari, akwai alamun ƙoshin endothelial. Muscleararrawar ƙwayar ƙwayar cuta mai laushi alama ce mai mahimmanci pathology na atherosclerosis. Wannan tsari yana motsawa ta platelet mitogen, wanda ya kasance an ɗan rarrabe shi. Ba a yi nazarin Mitogen a cikin ciwon sukari ba. Yawan tarawar lipids a fannin cutar atherosclerotic rauni na faruwa ne a mafi yawan nau'ikan cholesterol na ciki. A cikin ciwon sukari wanda ba a sarrafa shi ba, karuwar matakan lipoproteins mai yawa a cikin jini da rage matakan babban plasma lipoproteins mai yawa yana sanya ƙawancewar lipid a cikin manyan tasoshin.

Akwai tabbacin yanayin rashin lafiya a cikin wasu masu fama da cutar sankara. Tare, waɗannan abubuwan rashin daidaituwa na endothelial, platelet, m tsoka, lipoprotein da halayyar coagulation za'a iya la'akari dasu azaman taimakawa ga matsalar hanzarta atherosclerosis a cikin ciwon sukari. Cikakken fahimta game da pathogenesis na wannan tsari yana taimaka wajan ci gaban hanyoyin magance cututtukan da suka dace.

Cututtukan zuciya na cututtukan zuciya a cikin marasa lafiya da masu ciwon sukari

Ana tattauna batun atherosclerosis da ciwon sukari akai-akai kuma ana bincika su. A halin yanzu, ra'ayin cewa ciwon sukari na ba da gudummawa ga hanzarta atherosclerosis ya haifar da adadi mai yawa na sashe da ɗakunan asibiti da cututtukan cututtukan dabbobi.

An bincika gwajin maganin 50,000 kuma an gano cewa a cikin wannan rukuni guda (yawanci ya fi shekaru 40), mutuwar daga cututtukan ƙwayar cuta daga cututtukan ƙwayar cuta a cikin marasa lafiya da ciwon sukari mellitus an lura sau 2-3 sau da yawa fiye da a cikin mutane ba tare da cutar ba. A cikin marasa lafiya da ke fama da cutar sankarar mahaifa, 30.2% na lokuta an lura, a cikin marasa lafiya ba tare da ciwon sukari ba - 19.4%, rukunin kwatancen sun dace da shekaru.

A cikin binciken sashi na 416 na ciwon sukari, an sami karuwa sosai a cikin yanayin cutar atherosclerosis idan aka kwatanta da waɗanda basu da ciwon sukari a cikin rukunin shekaru sama da 40.

Binciken sashin bayanai

A cikin ƙungiyar shekaru 30-60, an gano cutar zazzabin cizon sauro a cikin kashi 82.2% na mutane. Mafi yawan cututtukan atherosclerosis a cikin 56,3%. Ba a ambaci babban bambanci a cikin abin da ya faru na atherosclerosis a cikin marasa lafiya da ke fama da rashin ciwon sukari yayin rayuwa ba a cikin shekarun ƙungiyar 50-55, 60-69 da shekaru 70-79. Amma canje-canje atherosclerotic a cikin aorta da na jijiyoyin jini a cikin marasa lafiya da ciwon sukari mellitus sun mamaye yanki mafi girma fiye da mutanen da ba su da su. Wani gagarumin karuwa a yankin da ke fama da cutar ta atherosclerotic plaque, marubutan sun gano a cikin 12 daga cikin marassa lafiya 32 da suka mutu sakamakon cutar sankarau a shekaru 50 da haihuwa.

Hanyoyin rigakafi a cikin atherosclerosis, musamman nau'in ciwon sukari na 2

Atherosclerosis da cututtukan zuciya da ke biyo baya (CVD) sune manyan rikice-rikice na ciwon sukari na type 2. Atherosclerosis cuta ce mai saurin kamuwa da cuta wanda ya haɗa da ƙwayoyin immunocompetent na nau'o'i daban-daban waɗanda ke cikin rauni. Kodayake kumburi da kunnawa na rigakafi na iya zama mafi ma'ana tare da atherosclerosis da nau'in ciwon sukari na 2, babu bambance-bambance masu mahimmanci tsakanin masu ciwon sukari da marasa ciwon sukari.

Don haka, dalilai masu kama suna da alaƙa a cikin rigakafi na rigakafi wanda ke hade da atherosclerosis a cikin ƙungiyoyi biyu. Ba a san musabbabin kunnawar rigakafi ba, kuma akwai hanyoyi da yawa wadanda ba sa iyakance ba. Oxidized da / ko enzymatically gyaran siffofin low lipoproteins mai yawa (OxLDL) da kwayoyin da suka mutu suna nan a cikin filayen atherosclerotic. OxLDL na iya taka rawa, kasancewa pro-inflammatory da immunostimulatory, tunda yana kunna ƙwayoyin T kuma yana cikin cytotoxic a cikin manyan taro.

Phospholipids mai kumburi a cikin OxLDL suna haɗuwa, tare da phosphorylcholine (PK) a matsayin ɗayan magungunan da aka fallasa.

Kwayoyin cuta da kwayar cutar sun tattauna game da yiwuwar haifar da kunnawar rigakafi, amma yana da wahala a sami hujjoji na kai tsaye don tallafawa wannan hasashe, kuma gwajin kwayoyin cuta a cikin mutane sun kasance mara kyau ko ba a yarda dasu ba. Hanyoyin kariya na zafi (HSPs) na iya zama ɗayan manyan abubuwan da ake neman martani game da rashin ƙarfi na rigakafi. Causesarin abubuwan da ke haifar da katsewa kai tsaye sun haɗa da cytokines kamar interleukin 1β (IL-1β), tumor necrosis factor (TNF), da masu shiga tsakani na lipid a cikin hanyar leukotrienes.

Bugu da ƙari, tare da ciwon sukari, hyperglycemia da damuwa na damuwa, a fili, suna hanzarta haɓakar atherosclerosis, ɗayan hanyoyin na iya zama haɓakar martanin rigakafi. Don tabbatar da cewa halayen rigakafi sune sanadin atherosclerosis da cututtukan zuciya, ana buƙatar ƙarin nazarin tare da maganin immunomodulating.

Ciwon sukari na 2 wani babban matsala ne da girma a duk faɗin duniya, kuma ba kawai a cikin ƙasashen da ake kira ci gaba ba. Baya ga cutar nephropathy da cututtukan ƙwayar cuta, cututtukan zuciya (CVD) da hanzarta atherosclerosis sau da yawa suna faruwa a cikin ciwon sukari, duka nau'in 1 da 2. Babban fifiko a cikin wannan bita shine kunnawa na rigakafi a atherosclerosis, musamman a cikin nau'in ciwon sukari na 2.

Dangantaka tsakanin nau'in ciwon sukari nau'in 2 da kumburi tana da kyau sosai, kuma akwai alamun ciwon kumburi a duka ciwon sukari da kuma jurewar insulin (IR), alama ce ta nau'in ciwon sukari na 2. Hakanan, tare da cututtukan atherosclerosis da cututtukan zuciya, kumburi mara nauyi shine babban fasali, kuma tare da atherosclerosis, an kunna ƙwayoyin rigakafi, kamar ƙwayoyin T da ƙwayoyin gabatarwar antigen, masu yawa.

Kodayake za a iya ƙara kumburi da girman ƙwayar necrotic tare da atherosclerosis a cikin ciwon sukari, babu wani bambance-bambance a cikin yawan macrophages, lymphocytes, da babban kumburi a cikin filaye ko ƙwayoyin atherosclerotic tsakanin masu ciwon sukari da marasa ciwon sukari, bisa ga binciken mafi girma a wannan yanki. Don haka, da alama babu wani bambanci na asali tsakanin kunnawar rigakafi da kumburi da ke cikin atherosclerosis tsakanin masu fama da cutar siga idan aka kwatanta da masu ciwon sukari. Koyaya, macrophages da thrombi na sama zasu iya tsawan lokaci bayan alamun ischemic a cikin ciwon sukari, wanda zai iya ƙara haɗarin sake dawo da cutar cututtukan zuciya a cikin wannan yanayin da abubuwan haɗari, tunda hyperglycemia ta halitta yana taka rawa ta musamman.

Wani mummunan tasirin kumburi yana tasowa ne daga mahangar juyin halitta, wata ila zai iya kiyaye kariya daga cututtukan cututtukan cuta da kuma gyara lalacewar nama, wanda shima rauni zai iya haifar dashi. Bayyanannun alamun cutar kumburi - zafi, kumburi, jan launi, zazzabi da rage aiki - an riga an bayyana su a cikin maganin Hippocrates.

Lokacin da ba a kawar da kumburi mai kumburi ba, amma a maimakon haka ya ci gaba kuma ya zama na kullum, zai iya zama matsala mai wahala.

Misalai na cututtukan kumburi da cuta sune cututtukan rheumatic, kamar:

• arthritis (RA),
• na tsari lupus erythematosus (SLE),
• atherosclerosis da babban sakamako, cututtukan zuciya, wanda ya haɗa da rauni na zuciya (MI), ciwo na jijiyoyin jini (ACS), lameness da bugun jini,
• Cutar Alzheimer
• nau'in ciwon sukari na 2
• haɓakawa a cikin IR har ma da kiba na ciki da osteoarthritis - suna da abubuwan haɗari.

Abubuwan haɗin gwiwa tsakanin waɗannan yanayin sanannu ne. Misali, ciwon sukari na 2 shine babban hadari ga cutar atherosclerosis da cututtukan zuciya (tare da shan sigari, hauhawar jini, dyslipidemia, shekaru da jinsi na maza). Cutar Alzheimer da atherosclerosis da / ko cututtukan zuciya suna da abubuwan haɗari da yawa, kuma shan taba sigari ne mai haɗari ga RA ban da sanannun sakamakon cutar cututtukan zuciya. Hakanan ya bayyana a sarari cewa akwai ƙungiyoyi tsakanin cututtukan rheumatic da atherosclerosis / CVD, musamman a SLE.

Hakanan a cikin RA, akwai karuwar haɗarin cutar cututtukan zuciya bisa ga rahotanni da yawa, kuma wani bincike-bincike na meta na kwanan nan ya ba da shawarar cewa atherosclerosis per se ya fi yawa a cikin marasa lafiya a RA. Yana da ban sha'awa a lura cewa akwai rahotanni waɗanda su ma suka bayyana haɓakar haɗarin kamuwa da cutar sukari nau'in 2 a RA.

Magungunan anti-inflammatory sun inganta tsinkayen marasa lafiya da yawa tare da yanayin kumburi, mafi yawan misalai sune abubuwan da suka shafi halittu kamar tumor necrosis factor inhibitors (TNF) inhibitors in RA da sauran yanayin autoimmune. Sabili da haka, akwai ainihin buƙatar kimantawa don yin maganin rigakafi da maganin rigakafi a cikin sauran yanayin kumburi.

Wataƙila mai ban sha'awa shine samfuran ƙwayoyin halitta, irin su inhibitors na TNF, na iya zama da warkewar tasiri a cikin atherosclerosis da ciwon sukari na type 2 da rikitarwarsu. Koyaya, wannan baiyi kama da kowane bangare ba. Kodayake ƙididdigar tsarin TNF yana da tasirin anti-cachectic a cikin marasa lafiya tare da RA, tasirin anti-TNF na IR bai dace ba, ya danganta da tsananin cutar da girman kumburi. Koyaya, wani rahoto game da kwanan nan ya ba da shawarar cewa magani tare da sabon hanawa na T-cell yana da tasiri mai ban mamaki akan IR a RA.

Oxidation da sauran gyare-gyare na LDL da sauran abubuwan haɗin

Poarancin lipoprotein mai yawa ana iya canza shi ta hanyar hadawan abu da iskar shaka da / ko kuma gyaran enzymatic na phospholipases, a matsayin misali guda. LDL kuma shine yawanci kasancewa a cikin kyallen kamar yadda intima na arteries, inda zai iya ɗaura zuwa matrix na proteoglycan, musamman bayan gyaran. An yi imanin cewa wannan ɗaurewar abu ne farkon abin da ya faru a cikin atherogenesis dangane da "martani ga kiyayewar" hypothesis.

Oxidized low yawa lipoprotein yana da pro-mai kumburi da immunomodulating kaddarorin, kunna endothelial sel, monocytes / macrophages da sel T. OxLDL shima mai guba ne a yawan taro, kuma muhimmin fasali na raunukan atherosclerotic, wataƙila ba'a ɗanɗana shi ba, shine adadin sel da suka mutu. Don haka, yana yiwuwa OxLDL shine ɗayan abubuwan da ke haifar da irin wannan mutuwar ƙirar sel. EnDymatically LDL na iya taka muhimmiyar rawa, kuma PLA2, yana haifar da irin wannan canjin, an bayyana duka a cikin jijiyoyin al'ada da kuma raunuka na atherosclerotic kuma suna iya haifar da kunna DC. Phospholipids mai illa, kamar lysophosphatidylcholine (LPC) da / ko platelet na kunnawa firikwensin (PAF), suna haifar da yawancin sakamakon OxLDL: s, wanda zai iya faruwa ta hanyar mai karɓar PAF ko wasu hanyoyin, ciki har da mai karɓar Toll-kamar, da kuma hulɗa mai karɓar karɓar karɓa .

Gabaɗaya, oxidized phospholipids (OxPLs) suna haɗuwa da sake kunnawar rigakafi a atherosclerosis kuma ana iya samun shi daga canjin LDL, amma kuma daga canje-canje a cikin membrane tantanin halitta. Irin waɗannan oxPL sun haɗa da LPC, kuma mafi yawan gajeren matsayi na sn-2 a cikin abun da ke cikin kitse mai acid yana aiki azaman tsarin haɗarin ƙwayoyin cuta (DAMP). Hadawan abu da iskar shaka yana canza OXL zuwa alamomin I masu canzawa wadanda masu karɓa da masu karɓa suke da karɓa, kamar su masu karɓa mai narkewa, abubuwan da ke faruwa a zahiri, da furotin na C-reactive (CRP). Babban jigo a cikin wannan tsarin daban shine watakila cirewar tsufa da ƙwayoyin matattu, amma kuma oxidized ko wasu abubuwan da aka inganta na lipoproteins.

Ana ba da shawarar Statins don ciwon sukari na 2 don rigakafin cututtuka na tsarin kewaya da ingantaccen aikin zuciya. Magunguna suna da sakamako mai kyau a cikin rage ƙwayar cholesterol.

Ana ba da shawarar Statins don ciwon sukari na 2 don rigakafin cututtuka na tsarin kewaya da ingantaccen aikin zuciya. Magunguna suna da sakamako mai kyau a cikin rage ƙwayar cholesterol.

Atherosclerosis da varicose veins sune cututtuka daban-daban. Yana da mahimmanci don tsara jigilar magani daidai da ƙayyade sashi - kawai ƙwararren likita ne zai iya wannan.

Jiyya na jijiyoyin bugun gini atherosclerosis ana yinsu ne da fahimta. Suna amfani da kwayoyi, magungunan jama'a, a wasu yanayi, maganin tiyata.

Bayyanar cututtuka na atherosclerosis da ciwon sukari na ciwon sukari

Lalacewa daga aorta da jijiyoyin jijiyoyin jini yana haifar da bambance bambancen cututtukan zuciya (rashin jin daɗi da yanayin jijiya), tare da rikitarwa:

• zuciya sabo
• tsananin rudani,
• maganin ƙwayoyin cuta na jijiyoyin jini,
• bugun zuciya
• rarrabuwa a cikin jini
• maimaita bugun zuciya
• kwatsam cardiac kama.

Canje-canje na atherosclerotic a cikin tasoshin kwakwalwa yana haifar da bugun jini ko encephalopathy dyscirculatory, ya danganta da yanayin ko cutar da take fama da ita, tare da hauhawar jini, hauhawar jini a cikin kwakwalwa sau da yawa.

Ana cire atherosclerosis na jijiyoyin ƙananan ƙarshen ana samun su a kusan ɗaya cikin biyar na masu fama da cutar sankarar bargo. Yana tattare da wannan alamomin:

• rage hankali
• numbance da tingling a cikin ƙafa,
• ƙafafu masu daskarewa koyaushe
• zafi lokacin tafiya (magana mai rarrabuwa) a cikin tsokoki na ƙasan kafa, femur da gluteus.

Tare da raguwa sosai a cikin zubar jini, mummunan nau'in ischemia na nama yana faruwa, yana faruwa ne daga cikin necrosis - necrosis da gangrene na ƙafa. Tare da ƙananan lalacewa - yanke, fasa, kamuwa da fungal - sannu a hankali yana warkar da cututtukan trophic.

Binciken yanayin jinin jini

A cikin atherosclerosis na gefe, ana buƙatar shawara tare da likitan jijiyoyin bugun jini, likitan zuciya yana bincika marasa lafiya tare da angina pectoris, da kuma ƙwararren mahaifa tare da alamun cerebral. Suna iya fadada jerin dakin gwaje-gwaje da hanyoyin nazarin kayan aiki. Mafi yawanci shawarar:

• gwajin jini ga glucose, haemoglobin, glycrol, triglycerides, hadaddun kwayoyin lipoprotein, coagulogram,
• ECG, saka idanu akan karfin jini da ECG bisa ga Holter, gwaje gwaje na aiki, duban dan tayi na zuciya, aorta, scintigraphy, coronarography, angiography of artron arteries tare da CT ko MRI,
• Duban dan tayi na jiragen ruwa na wuyansa da kai a yanayin binciken duplex, angiography of the cerebral arteries,
• Duban dan tayi tare da dopplerography of the arteries of the ƙananan ƙarshen, angiography of the jiragen of the kafa, rheovasography.

Jiyya na atherosclerosis na ƙananan sassan a cikin marasa lafiya

Ga marasa lafiya tare da cin zarafin mai da na lokaci guda tare da metabolism, ana amfani da rukunin kungiyoyin magunguna masu zuwa:

• rage yawan sukari na jini - kasancewar lalacewar yaɗuwa da jijiyoyin jiki da ƙarancin tasirin allunan alamu ne na insulin farji don kamuwa da ciwon sukari na 2, sannan na farko an wajabta hanyar tsananta hanyar sarrafa hormone,
• hypocholesterolemic - Lovastatin, Atocor, Liprimar a kan abinci,
• jami'ai na kwayan cuta - Clopidogrel, Dipyridamole, Ipaton, Aspirin,
• anticoagulants - Heparin, Clexane,
• inganta hawan jini - Berlition, Actovegin.
• antihypertensives (matsin lamba na matsin lamba a cutar sankara shine 135/85 mm Hg) - Prenesa, Kapoten, Lerkamen

Rage abinci idan kuna da matsaloli

Babban ka'idodin abinci mai gina jiki na asibiti don kamuwa da cutar atherosclerosis sune:

• karancin abinci - sau 5-6,
• raguwa cikin adadin kuzari mai yawa tare da nauyin jiki,
• ƙi abinci na gari da abinci mai daɗi,
• Yawan cin carbohydrates daga kayan lambu (ban da dankali), burodin baƙar fata, hatsi, 'ya'yan itãcen marmari (banda inabi, ayaba),
• low-mai nama ba fiye da sau 3 a mako, mafi yawanci samun furotin daga kifi, low-mai m-madara m, abincin teku,
• wariya daga menu na naman alade, rago, sausages, samfuran ƙare-ƙare da offal, broths nama, abincin gwangwani,
• tare da ƙara matsa lamba na sodium chloride ya kamata ba 5 5 a rana ba, a maimakon haka, an ba da shawarar bushewar ruwan teku, an murƙushe shi a cikin ɗanyen kofi, ganye da ruwan lemun tsami,
• don haɓaka fitowar cholesterol da sukari mai yawa, ana bada shawara ga burodi, ana tafasa su da ruwan zãfi kuma an haɗa su da hatsi, cuku gida, ruwan 'ya'yan itace, ana amfani da abinci don gurasa, kuma an shirya jita-jita na farko a kan kwanon,
• Zai fi kyau ku ci kayan lambu a cikin salatin da aka shirya tare da tablespoon na kayan lambu ko steamed, karas, beets da dankali ba a yarda ba sau 3 a mako,
• don shirye-shiryen abubuwan zaki suna amfani da 'ya'yan itatuwa da ba a girka su ba, maye gurbin sukari.

Kalli bidiyon ciwon siga:

Haɗin cutar ciwon sukari da atherosclerosis yana haifar da lalacewar manyan jijiyoyin jini da ƙananan, ƙananan tasoshin. Tare da raunin insulin, mai mai narkewa yana ƙaruwa, da yawaitar glucose yana lalata choroid, yana sauƙaƙe abin da aka makala daga cikin kayan masarufi.

Kuma a nan ne ƙarin game da arrhythmias a cikin ciwon sukari.

Macroangiopathy yana shafar jijiyoyin zuciya, kwakwalwa da jijiyoyin jijiyoyin jini. Don neman magani, ana yin magungunan ƙwaƙwalwar ƙwayar cuta. Da ake buƙata don rage yawan sukari na jini da cholesterol shine ingantaccen abinci mai gina jiki.

Abincin don atherosclerosis na tasoshin ƙananan ƙananan, kwakwalwa da zuciya sun haɗa da wariyar wasu nau'ikan samfura. Amma wannan dama ce ta rayuwa tsawon lokaci.

Idan ba zato ba tsammani lameness, jin zafi yayin tafiya, to waɗannan alamu na iya nuna lalata atherosclerosis na tasoshin ƙananan ƙarshen. A cikin yanayin ci gaba na cutar, wanda ya wuce cikin matakai 4, ana iya buƙatar motsi. Wadanne zaɓuɓɓukan magani ake samu?

Masu ciwon sukari na cikin hadarin kamuwa da cututtukan zuciya. Samun infasa daga cikin cututtukan zuciya na iya haifar da mutuwa. Ajiyar zuciya yayi mai sauri. Tare da nau'in 2, barazanar ta fi girma. Ta yaya jiyya zata gudana? Mecece kayan aikinta? Wace irin abinci ake buƙata?

Wuraren da aka gano daga cikin kwalaben carotid artery na da matukar hadari ga kwakwalwa. Jiyya yakan shafi tiyata. Cire ta wasu hanyoyin na iya zama da inganci. Yadda za a tsabtace tare da rage cin abinci?

Ischemic bugun jini yakan faru a cikin tsofaffi sau da yawa. Sakamakon haka bayan shekaru 55 suna da matukar wahala, murmurewa yana da wuyar ganewa kuma ba koyaushe yake nasara ba, kuma hasashen ba shi da fata. Cutar kwakwalwa mai wahala a gaban ciwon suga.

Ba haka ba ne mai ban tsoro ga lafiyar mutane, arrhythmia tare da ciwon sukari na iya zama babbar haɗari ga marasa lafiya. Yana da haɗari musamman ga ciwon sukari na 2, saboda zai iya zama sanadiyar bugun jini da bugun zuciya.

Idan an gano cutar angiopathy, magungunan jama'a sun zama ƙarin hanya don rage lokuta marasa kyau da hanzarta jiyya na baya. Hakanan zasu taimaka tare da maganin ciwon sukari, cututtukan atherosclerotic angiopathy.

Gabaɗaya, ƙwaƙwalwar ƙwayar cuta ta Menkeberg tana kama da cututtukan cututtukan ƙwayar cuta na mahaifa. Koyaya, cutar ta bayyana ta hanyar kwatancen ganuwar, kuma ba ta hanyar ajiyar cholesterol ba. Yaya za a bi da Menkeberg arteriosclerosis?

A lokaci guda, ciwon sukari da angina pectoris suna haifar da mummunar haɗari ga lafiyar. Yaya za a bi da angina pectoris da ciwon sukari na 2? Wace irin rikicewar zuciya zata iya faruwa?

A pathogenesis na atherosclerosis a cikin ciwon sukari

Atherosclerosis shine mafi yawan cututtukan jijiyoyin jiki. Maganin etiology, shine, sanadin atherosclerosis a cikin nau'in ciwon sukari na 2 na mellitus, ya zama cin zarafi a cikin ayyukan metabolism na lipid. Rashin insulin dangi na haifar da karancin glucose da mai mai yawa daga jini.

Kwayoyin jikin mutum suna fama da yunwa kuma hanta tana ɓoye abubuwan gina jiki a cikin ƙari na ƙarin ƙwayar lipids, wanda, sake, ba kyallen takama da ƙwaƙwalwa, saboda haka akwai matakan ƙaruwar ƙwayar lipids a cikin jini, yawan abin da aka ajiye a cikin jiragen ruwa. Kuma mafi girman matakin sukari na jini, da saurin aiwatar da wannan tsari. Atherosclerosis a cikin ciwon sukari yana haɓaka kimanin shekaru 8-10 cikin sauri.

Marasa lafiya suna da saurin kamuwa da cututtukan zuciya, wato angina pectoris, gajiyawar zuciya, suna da iskar myocardial sau 2-3 sau da yawa, da kuma bugun jini sau 1.5-2 sau da yawa. Wani mummunan rikicewa shine cutar zazzabin cizon sauro a cikin mahaifa na baya, wanda ciwo ya bayyana a kafafu, a yanayin gaba wannan shine mafi yawan sanadin kafa. A sakamakon haka, raunuka sun haɗu a cikin gabobin da ke gaba:

Sakamakon atherosclerosis akan ci gaban ciwon sukari

Abubuwan da ke haifar da atherosclerosis a cikin ciwon sukari a bayyane suke, amma shin akwai wata amsa? Sau da yawa a cikin mutane masu cutar atherosclerosis, ana gano cutar sukari nau'in 2, amma wannan ba wai kawai saboda na biyu ne sanadiyyar ta biyu. A bango daga kiba, ana kuma lura da cholesterol jini. Kuma kiba yana haifar da cin zarafin metabolism. Sau da yawa mutane masu kiba suna fama da hauhawar jini, wanda kuma yana haɓaka lalacewar jijiyoyin jiki. Don haka, allunan atherosclerotic a cikin ciwon sukari na iya zama a gabanin ci gabanta.

Lokacin da filaye ke toshe ƙwayar jirgin ruwa sama da kashi 70%, gazawar jijiyoyin jiki a cikin sashin da ke faruwa, ƙwayoyin suna karɓar iskar oxygen, kuma idan sukari ya lalata, sel kuma suna rasa glucose. Wannan yana haifar da rashin abinci mai gina jiki, musamman, ƙwayoyin jijiya, raunin jijiyoyin haɓaka. Wannan kuma yana haifar da haɗarin rikitarwa masu haɗari - lactic acidosis, mutum na iya fada cikin rashin lafiya.

Jiyya na atherosclerosis a gaban ciwon sukari mellitus

An ba da kulawa sosai game da cutar atherosclerosis a cikin ciwon sukari. Kwayoyi masu narkewa a cikin jini ana gyara su ta hanyar kwayoyi daga rukunin Statin. Matsayi na cholesterol ya kamata ya zama ƙasa da na al'ada. Sabili da haka, an wajabta gumaka ga duk masu haƙuri. Idan tasirin su bai isa ba - suna ƙara wasu magungunan rage ƙwayoyin cholesterol. An tsara masu tunani na jini don hana ƙwanƙwasa jini a kan filayen atherosclerotic.

Daga cikin magungunan da ke rage karfin sukari na jini, idan babu contraindications, an wajabta Metformin, ana iya haɓaka shi da sauran magunguna da insulin. Magungunan rigakafin ƙwayoyin cuta suna magance matsin lamba a tsakanin ƙasa da 130/80 mm. Hg. Art. Wajibi ne a bar shan sigari da barasa, motsa jiki, daidaita nauyi. Ba shi yiwuwa a kawar da filayen da ke tasowa a bangon jijiyoyin jini tare da magunguna kuma a cikin matakai na ci gaba ana buƙatar likita na jijiyoyin bugun jini.

Abincin ya hada da kin amincewa da duk wasu zartarwar kayan abinci, kayan gari da maye gurbinsu da hatsi, mafi dacewa buckwheat, oatmeal.Kowace rana kuna buƙatar cin 400 grams na kayan lambu da 'ya'yan itatuwa, sai dai dankali, ayaba, innabi, ɓaure,' ya'yan itatuwa da aka bushe. Atsan dabbobin: man, nama mai ƙima, man alade - maye gurbin kayan lambu. Aƙalla sau biyu a mako, kuna buƙatar cinye kifin marine (mackerel, herring, da sauransu).

Rashin narkewar sukari koda yaushe yana tare da atherosclerosis. Kuma tare da na ƙarshen, ana yawan gano cutar sukari. Suna cutar da juna. Sabili da haka, don rigakafin, ya wajaba don yin yaƙi da wuce haddi kuma jagorantar rayuwa mai kyau. Kuma tare da haɓaka cutar, bi umarnin umarnin likita don hana rikitarwa masu haɗari.

Me yasa ilimin cututtukan jijiyoyin jiki ya bayyana?

Tun lokacin da aka gano ƙwayar cutar atherosclerosis, likitoci sun yi nasarar kirkiro jerin abubuwan da ke haifar da haɓakar wannan cutar. Wadannan sun hada da:

• rage cin abinci mai amfani da abinci (yawan amfani da kitse, soyayyen abinci na asalin dabba, kayan abinci, burodi, da sauransu),
• rashin motsa jiki
• shan taba sigari,
• maganin cututtukan jini da cututtukan zuciya,
• tsawan wahala.

Jikin ɗan adam tsari ne mai ban mamaki wanda ke da ikon yin tsayayya da yawancin tasirin tasirin waje. Abin da ya sa har ma abubuwan da ke sama ba koyaushe ba suke haifar da faruwar cutar atherosclerosis. Don fara aiwatar da cututtukan jijiyoyin a cikin tasoshin, dole ne a cika ƙarin yanayi biyu - matakin haɓaka kullun da keɓaɓɓen cholesterol da lalacewar ciki ga tsarin jini "manyan hanyoyi".

Af, 'yan likitoci, da marasa lafiya da kansu, suna yin la’akari da gaskiyar lalacewar injin ɗin jijiyoyin jini. Pressureara yawan matsin lamba wanda ke ci gaba na dogon lokaci yana haifar da bayyanar microcracks da ƙananan ruptures na capillaries da arteries. A waɗannan wurare ne adadi mai tsoka mai ɗorewa ya fara tsayawa. Tsarin tasoshin ya zama mafi rauni, elasticity ya ɓace. Daga qarshe, cikin shekaru masu yawa, plaque mai dauke da sinadarin cholesterol bai rufe hanyar jini ba, har ila yau yana rage kwararar jini.

Yawancin marasa lafiya da aka gano tare da atherosclerosis da ciwon sukari mellitus suna da haɓakar thrombosis.

Ta yaya ake nuna cututtuka biyu?

Atherosclerosis yana haɓaka ciwon sukari na mellitus tare da ƙananan bambance-bambance na waje. Ya danganta da abin da jinin jini ya shafa da tasirin cholesterol, alamomin gaba ɗaya zasu canza. Mafi sau da yawa, ƙwayoyin atherosclerotic suna zaune akan bangon manyan jijiyoyin jini waɗanda ke ba da jini ga kwakwalwa, zuciya, da ƙananan ƙarshen. Misali, tare da lalacewar cututtukan aorta da na jijiyoyin jini, mara lafiya yana da hatsarin haɓakar bugun zuciya da cutar jijiyoyin jini, alamun angina pectoris, jin zafi a cikin sternum ya bayyana.

Idan halittun cholesterol sun zauna a kowane bangare na brachiocephalic arteries, to kwayar kwakwalwa ta shafi da farko. Mai haƙuri ya fara jin ciwon kai, tsananin farin ciki (suna nuna yunwar oxygen na jiki), kuma idan ba a kula da shi ba, cutar na iya haifar da ci gaban bugun jini. Rashin nasarar tasoshin jini na ƙananan ƙarshen daga waje yana bayyanar da kanta a cikin canji cikin launi da zafin jiki na fata, haɓakar ƙoshin trophic, bayyanar yanayin halayen, da kumburi mai taushi.

Atherosclerosis na ƙananan sassan na ƙarshe na iya buƙatar maganin tiyata, watau yanki. Likitocin suna amfani da irin waɗannan matakan ne kawai a lokuta na musamman, lokacin da mai haƙuri yana da girma kuma mai nuna ƙoshin ƙwaƙwalwar nama.

Ciwon sukari na atherosclerosis yana wahalar da yanayin ta dalilin rashin insulin, aikin sarrafa kitsen dake jikinsa ya rikice. Abun haɗin yana haɓakawa a hankali kuma a cikin ƙaramin adadin, matakin babban lipoproteins mai yawa yana fara raguwa tare da karuwa a cikin ayyukan abubuwa tare da ƙarancin ƙarancin girma. Abin da ya sa mai haƙuri ba kawai matsaloli tare da sukari na jini ba, har ma da cutar atherosclerosis tana taɓarɓarewa.

Yaya za a magance cutar?

Yaƙi da atherosclerosis da ciwon sukari dole ne a kusanci su tare da kulawa. Ba za mu ba da sunayen takamaiman magunguna ba, tunda likitan halartar ya kamata ya tsara ta da likitan halartar yin la'akari da shaidar haƙuri. Idan zamuyi magana game da magance cututtukan guda biyu a takaice, to yakamata ya zama haka:

1. An tsara lafiya sosai don rage sukarin jini. Likita, yin la’akari da sakamakon bincike na asibiti da na kayan aiki na mai haƙuri, yakamata a tsara daidai yawan magungunan da zasu taimaka rage matakan glucose, da rage yiwuwar lalata bangon jijiyoyin jini.
2. Gyara rayuwa. Dukkanin marasa lafiya da likitan halayyar endocrinologist an wajabta musu abinci na musamman. Tare da taimakonsa, marasa lafiya ba kawai suna gwagwarmaya tare da ƙarin fam ba (kowane mutum na biyu da ke da ciwon sukari yana fama da kiba), amma kuma yana nuna alamun glucose a matakin al'ada. Tare da atherosclerosis, halayen cin abinci na gari na iya rage cholesterol da yawan adon lipoprotein akan bangon jijiyoyin jini.
3. Cire munanan halaye. Shan taba da yawan shan ruwa na iya yin magani ga atherosclerosis da ciwon sukari mara inganci. Taba sigari yana shafar tsarin zuciya da jijiyoyin jini, yana haɓaka ragowar tasoshin jini. Giya na iya haifar da tsalle tsalle a cikin sukarin jini, wanda hakan zai cutar da yanayin mara lafiyar. Ka'idodi masu kyau kawai zasu taimaka inganta haɓaka da tsawon haƙuri na haƙuri.
4. Ikon hawan jini. Marasa lafiya suna buƙatar saka idanu akan karatun hawan jini a hankali. Ta hanyar riƙe su a matakin al'ada, marasa lafiya suna rage haɗarin mummunan tasirin cututtukan cututtuka akan mahimman gabobin - zuciya, kwakwalwa, hanta, ƙoda, da sauransu.

A ƙarshe, dole ne a faɗi cewa ganowar lokaci da kuma farawa don atherosclerosis, wanda aka haɓaka da asalin ciwon sukari, zai ba da haƙuri haƙuri da ƙarfin aiki, kiwon lafiya har ma da rayuwa. Karku daina zuwa wurin likita a cikin akwati mai tsawo kuma ku lura sosai da mahimman alamun alamun jikin ku!

Ta yaya ake kafa atherosclerosis a cikin ciwon sukari?

A yadda aka saba, harsashi na ciki na jijiya yana samar da sinadarin nitric oxide da abubuwa wadanda ke hana shigar kitsen kitse a ciki, adon jikin ruwan platelet. A cikin ciwon sukari mellitus, damar yin tsayayya da masu wuce gona da iri a cikin jiki an rage sosai saboda yawan glucose, cholesterol a cikin jini, hawan jini da halayyar halayen kumburi.

Sakamakon haka, haɗin tsakanin sel na jijiyoyin bugun jini ya raunana, saboda haka sun ƙona kitse a ciki. A can ne suka zama cibiyar samar da filayen atherosclerotic. A wurin mai adon mai, bangon jijiya yana samun sassauƙa, tsagewa da rushewa. Filatoci suna hanzarta zuwa wurin lalacewar jirgin ruwa, suna haifar da gudan jini (ƙwanƙwasa jini).

Atherosclerosis na ƙananan sassan

Tare da haɓaka filayen hannu, iyawar ƙwayar jijiyoyin a hankali ba su da matsala; ita kanta ta zama dattako saboda canjin ƙwayoyin salts a cikin ta. Don haka akwai rikice-rikice na wurare dabam-dabam - angina pectoris, encephalopathy diskila (lalacewar arteries), ciwo mai tsinkayewar magana.

Idan ƙwaƙwalwar ƙwayar cuta ba ta zama m ba (wanda aka fi dacewa a cikin ciwon sukari na mellitus), to farjinta ya lalace. An canza barbashi tare da gudanawar jini zuwa gabobin ciki, kwakwalwa, reshe. Wannan yana haifar da ciwon zuciya, bugun jini, da ɓacin ƙafa na ƙafa.

Kuma a nan akwai ƙarin game da raunuka a cikin ciwon sukari.

Babban kwalakwala mai yawa

Don canzawa kwayar cholesterol, an samar da hadaddun halittu wanda za'a ɗaura su da sunadarai. Idan akwai furotin gaba daya a cikin lipoprotein, to sai su daure mai mai da karfi. Irin waɗannan nau'ikan sufuri suna da yawan gaske kuma suna da tasiri mai kariya akan tasoshin jini. Ana kiransu "kyakkyawan cholesterol." A cikin ciwon sukari, yawansu yana ƙasa da na al'ada.

A cikin hadaddun ƙarancin ƙasa mai ƙima da rashin ƙarfi, mai mai ƙiba fiye da furotin, suna sauƙaƙa "rasa" cholesterol kuma sune manyan mahalarta aikin ginin filaye. “Fats mara kyau” suna cinye masu ciwon sukari. A lokaci guda, fasali mai mahimmanci shine cewa irin waɗannan canje-canje a cikin jini ba cikakken tunani bane na matakin glucose. Sabili da haka, akwai yanayi yayin da taimakon insulin ko allunan da mai haƙuri ya sarrafa don kawo sukari kusa da al'ada, kuma mai metabolism yana raguwa.

Levelsarancin ƙananan ƙwayoyin cholesterol masu yawa, haɓaka matakin ƙananan hadaddun ƙarancin lamuran jiki da ƙarancin matsakaitaccen ƙwayar triglycerides a cikin jini suna kamanni da cewa ana kiransu da ciwon sukari mai narkewa, ko dyslipidemia mai ciwon sukari. A cikin abin da ya faru, jurewar insulin yana taka rawa sosai.

Matsayi na glucose da insulin a cikin metabolism mai

Babban dalilin duk matsalolin ciwon sukari shine karancin insulin. Ko dai ana yin sa ta hanyar da ta dace (nau'in cuta ta 1), ko kyallen takarda basu amsa ba (nau'in 2). Wannan yana rushe aikin glucose a cikin tantanin halitta. Irin wannan yunwar tana haifar da cin zarafi:

• Ana amfani da sunadarai da kitse don samar da makamashi,
• sinadarai mai guba suna tarawa saboda rashin aiki na rayuwa,
• Akwai ƙarin dogaro da wadatar iskar oxygen (madadin hanyoyin da suke da iskar oxygen),
• an saki homonin damuwa (adrenaline, cortisol), mai hana jijiyoyin jini jini.

Acids da aka kirkira sakamakon rushewar nama na adipose suna amfani da hanta don gina kitsen nasu - triglycerides da cholesterol. Saboda haka, koda babu mai mai mai ne, ana samarwa da yawa a cikin jiki.

Muhimmancin Angiopathy

Ginin jirgin ruwa na mai haƙuri tare da ciwon sukari yana haɓaka da ƙima mai ƙarfi, ƙarancin nutsuwa da rashin iyawa. Hanyoyin ƙwayoyin hanzari suna amsawa da sauri ta hanyar taƙaita sigina na tsarin juyayi mai juyayi da canje-canje a cikin abubuwan da ke cikin jini, amma a hankali kuma ba faɗaɗa gabaɗaya. Babban hauhawar jini wanda ke kama da mai ciwon sukari yana bayar da gudummawa ga lokacin farin ciki na tsokar jijiya.

Addara wannan wannan shine raguwa gaba ɗaya na gudanawar jini saboda rauni zuciya da rauni na ƙananan tasoshin. Saboda haka, marassa lafiya suna da duk yanayin yanayin cututtukan atherosclerotic raunuka na jijiya, hanzarta ƙwayoyin jini, da cututtukan jijiyoyin bugun zuciya.

Rashin jin daɗi

Lalacewa ga ƙwayar zuciya

Ragewar zubar jini (ischemia) ta hanyar jijiyoyin jijiyoyin zuciya yana haifar da yawan jin zafi a cikin zuciya. Sakamakon gaskiyar cewa ana shafar fiber na jijiya a lokaci guda, halayyar halayyar bazai yiwu ba. Sabili da haka, an gano angina pectoris a ƙarshen mataki, ko mai haƙuri yana da bugun zuciya ba tare da gunaguni na musamman game da aikin zuciya ba. Hakanan hanyarsa na ɓoye, mara jin zafi.

Rashin magani a cikin mummunan lokacin yana haifar da rikice-rikice masu wahala, mutuwa ko jiyya na tsawan lokaci, dawo da sashi, raunin jijiyoyin jiki.

Cerebral arteriosclerosis

Mafi sau da yawa, carotid artery ya zama "makasudin" don ƙirƙirar ƙwayar cuta ta atherosclerotic. An kafa dogaro da rauni a kan saukan ruwa a matakin insulin a cikin jini. A lokaci guda, duka biyu masu karamin karfi (nau'in ciwon sukari 1 da ciwon sukari mai cinye 2 na insulin) da kuma babban mataki (matakan farko na nau'in 2, allurai na injections) suna taka rawa.

An lura da alamun masu zuwa a cikin marasa lafiya:

• ƙwaƙwalwar ƙwaƙwalwar ajiya
• low tunani da aikin yi,
• wahalar tattarawa da koyan sabon bayani,
• rikicewar damuwa, rikitarwa wajen daidaitawa,
• rashin tsaro lokacin tafiya.

A cikin maganganun ci gaba, encephalopathy na ciwon sukari yana haifar da raguwar hankali, canje-canjen halaye da rikice-rikice na tunani. Tare da matsanancin toshewar jirgin ko kuma lalatawar jijiya, bugun jini ya faru. A cikin ciwon sukari, ana nuna shi ta hanyar lalata halayen neurons da jinkirin dawo da ayyukan da suka ɓace (motsi, hankali, magana).

Canje-canje a cikin tasoshin ƙananan ƙarshen

Atherosclerosis obliterans (wanda ke faruwa tare da toshewa) ana bayyana shi ta hanyar jin zafi a farkon lokacin motsa jiki. Bayan doguwar tafiya, yakamata a dakatar da marasa lafiya domin jinya a cikin kafafu ya shuɗe. A hutawa, jin sanyi, narkewa da kuma tingling a cikin kafafu, sananne mai sauƙaƙewa tsokoki.

A tsawon lokaci, nisan da zaku iya tafiya ba tare da ciwo ba yana raguwa, kuma sakamakon rashin abinci mai gina jiki yana faruwa akan fatar - bushewa, bawo, fashe, rauni. Yin haɓakar raguwa mai mahimmanci a cikin zubar jini shine gangrene (necrosis).

Hadarin haɗakar ciwon sukari da atherosclerosis

Rashin lafiyar jijiyoyin jiki tare da haɗuwa da yawa na glucose da cholesterol ana ɗaukar su ta hanya mai tsanani. Marasa lafiya daga ƙungiyoyi masu haɗari suna iya kasancewa da rikice-rikice. Yawancin sakamako masu illa suna faruwa tare da:

• kiba, musamman tare da yawancin adon mai a ciki,
• tsawanta hawan jini ko canje-canjenta masu kaifi, rikice-rikice masu hauhawar jini,
• hali na thrombosis,
• da shekaru mutane daga 45 years old,
• menopause a cikin mata,
• sutudiyyar rayuwa
• ɗaukar nauyi na gado,
• shan taba
• yawan abinci mai guba, mai mai yawa a cikin abincin,
• hanyoyin kumburi na kullum.

Ga zuciya da kwakwalwa

Hanyar rashin daidaituwa na rashin aiki na zuciya (rashin jin zafi) yana halin cigaban rikice-rikice:

• yaduwar jini da yaduwar jini a cikin huhu, hanta, harin asma, huhun hanji,
• hatsarori masu haɗari na kari na rikice-rikice, har zuwa kamo bugun zuciya kwatsam,
• daga raguwar karfin jini, dakatarwar fitowar fitsari (bugawar zuciya),
• jini clots a cikin zuciya yayin atrial fibrillation da motsi a cikin kwakwalwa (hadarin bugun jini),
• samuwar sabon cuta (tsotsewar zuciyar zuciya) da katsewar myocardium,
• maimaita bugun zuciya.

Tare da matsanancin toshewar jirgin ruwa wanda ke ciyar da kwakwalwa, bugun zuciyar ischemic na faruwa. Thearfin mai haƙuri ya motsa kansa ba tare da tafin kafa ba, hasara a cikin yankin da abun ya shafa yana raguwa, magana, haɗiyewa, da hangen nesa sun lalace. Tare da zubar jini daga wani jirgin ruwa da ya lalace, alamomi na asarar hankali yana haɓaka da sauri, sau da yawa coma, da kuma ƙwayar cuta ta hanji tare da sakamako mai mutuwa.

A cikin rayayyun marassa lafiya, yana yiwuwa a haɗu da cututtukan ɓangaren hanji da na huhu, matsanancin rauni da guban jini (sepsis). Sau da yawa, murmurewa kawai sashi ne, mai haƙuri ya zama mai rauni, gaba ɗaya yana dogara da taimakon waje.

Yankunan ƙananan ƙananan

Sharpuntataccen ƙuntatawa na kwararawar jini zuwa ƙafa yana haifar da lalata nama tare da samuwar gangrene. Wannan yanayin yana da haɗari ga rayuwa kuma yana buƙatar yanke hannu don kulawa dashi. A cikin lamari na yau da kullun, ƙaramin rauni ko rauni na iya zama asalin tushen samuwar cutar sankarar mahaifa. An nuna shi ta hanyar warkarwa mai saurin motsawa, hali don ƙoshinta.

Yaduwar kamuwa da cuta mai zurfi cikin kyallen takarda yana haifar da osteomyelitis (kumburi da kasusuwa kashi), sepsis.

Jiyya na atherosclerosis a cikin ciwon sukari

Tare da cin zarafin lokaci guda na carbohydrate metabolism da rauni na atherosclerotic na arteries, ya wajaba don daidaita abinci mai gina jiki:

• ware abinci mai kyau a cikin cholesterol (mai kitse, mara kyau, sausages, samfurori da aka gama, mai dafa abinci, kayan kiwo tare da mai mai yawa, man shanu),
• iyakance nama zuwa sau 2-3 a mako, barin mai,
• bi shawarwari kan yawan abincin carbohydrate, cire Sweets da gari, nau'ikan 'ya'yan itatuwa da kayan marmari masu dadi daga abincin,
• maye gurbin gishiri a tebur tare da babban taro na potassium (misali, Himalayan ruwan hoda), amfani da ruwan 'ya'yan lemun tsami, ganye, abinci ba tare da gishiri don inganta dandano ba,
• aƙalla sau biyu a rana ku ci sabon salatin kayan lambu wanda aka girka tare da mai kayan lambu,
• yi kayan lambu (sai dai dankali) tushen menu,
• a matsayin tushen furotin na amfani da kifi, abincin abincin teku, mara mai mai mara mai mai wanda ba shi da kitse, farin kwai,
• don dafa abinci amfani da hawan iska ko kuma stewing cikin ruwa.

Magungunan ƙwayar cuta ya haɗa da rukuni na gaba na ƙwayoyi:

• insulin - idan ba zai yiwu a cimma matakin da ake buƙata na glucose a cikin jini ba, tare da mai haƙuri na 1, ana tura su zuwa cikin tsarin kulawa mai ƙarfi, kuma tare da nau'in 2, ana ƙara allurar hormone a cikin allunan,
• don rage yawan cholesterol - Atokor, Krestor,
• inganta hawan jini - Clexane, Asfirin, Plavix,
• daidaituwa na wurare dabam dabam na jini - Actovegin, Espa-lipon,
• angiotensin yana canza tsofaffin masu hana enzyme - Prestarium, Kapoten.

Idan ba zai yiwu ba a cimma nasarar da ake so tare da abinci da magunguna don rage ƙwaƙwalwar ƙwayar cuta, da kuma haɗarin cutar cuta mai narkewa, ana iya ba da shawarar tiyata.

Aiwatar da irin waɗannan hanyoyin:

• kau da atherosclerotic plaque,
• shigar da tsinkaye (firam ƙarfe) a cikin kunkuntar sashi,
• ƙirƙirar iska (kewaye) don gudanawar jini.

Kuma anan shine karin bayani game da ciwon sukari da angina pectoris.

Bayyanuwar atherosclerosis a cikin mai haƙuri tare da ciwon sukari sakamakon asali ne na rashin insulin. Babban matakan glucose da ƙarancin lipoproteins mai yawa yana haifar da yanayi don lalata bango na jijiyoyin bugun gini da samuwar plaques. A cikin tsari na yau da kullun, patency yana raguwa a hankali, kuma a cikin m akwai wani mummunan tashin zuciya (bugun zuciya, bugun jini, gangrene).

Don hana rikice-rikice da ci gaban lalacewa da jijiya, an tsara abinci da magunguna, kuma idan ba su da tasiri, ana tsara aikin ne.

Bidiyo mai amfani

Kalli bidiyon kan cutar sankara da kuma atherosclerosis:

Sau da yawa, ciwon sukari da angina pectoris suna damuwa lokaci guda. Fasalin na biyu ya bayyana tare da ciwon sukari fiye da sau da yawa, tunda marasa lafiya suna da matsala tare da tasoshin jini da jijiyoyin jini, ƙoshin jijiya. Zuciya ba ta rauni koyaushe, saboda mutane da yawa ba su da lokaci don fara magani a kan lokaci.

Sakamakon canje-canje a cikin jiki, sau da yawa marasa lafiya suna haɓaka ciwon sukari da hawan jini a lokaci guda. Ba kowa ba ne zai iya canza abincinsu, ya ci abinci, wanda ke haifar da buƙatar shan kwayoyi. Yadda ake shan kwayoyin hana kumburi da ciwon suga? Shin Taurine zai taimaka?

Idan mai haƙuri yana da cholecystitis da ciwon sukari a lokaci guda, to lallai zai sake tunanin abincin, idan cutar ta farko ta haɓaka. Dalilan da ya faru ya ta'allaka ne da yawan insulin, giya da sauransu. Idan muguwar ƙwayar cuta mai ƙwaƙwalwa ta ci gaba tare da ciwon sukari na mellitus, ana iya buƙatar tiyata.

'Yan kadan daga cikin marassa lafiya suna fuskantar irin wannan cuta kamar bugun zuciya da cutar sankarau. Yana faruwa duka a nau'in 1 da nau'in 2. Babban mace-mace a tsakanin marasa lafiya, nakasa ba a cire shi. Abincin bayan bugun zuciya yana taimakawa wajen dawo da jiki.

Sakamakon canji a cikin matakan sukari da kuma nunawa ga bangon insulin, raunin da ya shafi ciwon sukari ya bayyana wanda bai warke sosai. Waɗannan sune manyan dalilan da yasa fatar ta karye kuma ba ta da lafiya. Raunin kumburi mai purulent, mara warkarwa, kuka. Akwai kayan aikin musamman don magani da magani, wani lokacin ana buƙatar taimakon mai tiyata.