Babban mahimmancin hanyoyin maganin ciwon sukari sune:

- amfani da kwayoyi

- dosed aiki na jiki,

- ilimi mai haƙuri da kamun kai (makarantar sankara),

- Yin rigakafi da magani na ƙarshen rikice-rikice na ciwon sukari.

Manufar kula da ciwon sukari shine cimma cutar Normoglycemia, watau biyan diyya.

Marasa lafiya masu ciwon sukari dole ne su ware gaba ɗaya don amfani da sukari, syrups, adanawa, ruwan juji, kayan ledo, biscuits, ayaba, inabi, kwanan wata, barasa da wasu kayayyaki.

Jiyya tare da maganganu na baki hypoglycemic.

Magungunan da ake amfani da su na yau da kullun ana amfani da su zuwa kashi biyu: manyan sulfonylureas da biguanides.

Hanyar aiwatar da kwayoyi sulfonylureas hadaddun kuma saboda aikin tsakiya da na yanki. Sakamakon su na tsakiya a kan tsibirin na pancreatic an yi bayani ne ta hanyar motsawar insulin, haɓakawa a cikin ƙwayar ƙwayar клеток zuwa glycemia, wanda a ƙarshe yana haifar da ci gaba a cikin ɓoye insulin.

Extraarin-pancreatic sakamako yana haifar da karuwa a cikin amfani da glucose a cikin hanta da tsokoki tare da haɓaka samuwar glycogen a cikinsu, i.e. fitowar glucose daga hanta yana raguwa kuma tasirin aikin inzalin insulin yana ƙaruwa.

Biguanides perara yawan amfani da glucose na gefe a gaban insulin, rage gluconeogenesis, ɗaukar glucose a cikin ƙwayar gastrointestinal, da kuma rage ƙarancin insulin da ke cikin ƙwayar jini na marasa lafiya tare da kiba da nau'in ciwon sukari na 2. Bugu da kari biguanides da wani sakamako na nakuda. Dogon amfani da su tabbatacce yana tasiri metabolism na lipid (rage ƙarancin cholesterol, triglycerides).

Lokacin da sakamako mara amfani na jiyya tare da magungunan baka na hypoglycemic magunguna an wajabta maganin insulin.

Manyan janar An tsara wa marasa lafiya masu ciwon sukari don insulin: 1) nau'in 1 na ciwon sukari, 2) ketoacidosis, ciwon sukari, 3) asarar nauyi mai nauyi, 4) faruwar cututtukan cutuka, 5) tiyata, 6) ciki da lactation, 7) rashin sakamako daga amfani da wasu hanyoyin jiyya.

Tsarin insulin

Ta tsawon lokaci insulins su ne:

gajarta aiki - farkon farawa bayan mintuna 15-30, matsakaita na tsawon awa 5-8,

matsakaici tsawon - farawa na aiki bayan sa'o'i 1.5 -3, tsawon - 12-22 hours,

tsawan - farkon fara aiki bayan sa'o'i 4-6, tsawon lokaci - daga 25 zuwa 30 (36) awanni.

bovine (insulrap, ultralong, ultlente, da sauransu),

alade - mafi kusanci ga mutum, ya bambanta a cikin amino acid ɗaya (monoinsulin, actrapid, insulrap SPP, da sauransu),

bovine alade (gidantin-na yau da kullun, insulin-B),

ɗan adam - wanda aka samo ta hanyar injiniyan ƙwaƙwalwa daga E. coli da yisti mai yisti (humulin, monotard, protofan NM).

Da digiri na tsarkakewar insulin (daga somatostatin, polypeptide na pancreatic, glucagon, da sauransu):

na al'ada (na al'ada) - adadin abubuwan rashin amfani na iya zama har zuwa 1%, wanda ke ƙayyade babban immunogenicity,

monopic (Semi-tsarkake) - abubuwan ƙazanta sun ƙunshi kusan 0.1%,

monocomponent (tsarkakakku) - duk ruɗanin ɗan adam.

Monopic da monocoponent insulins sun fi tasiri fiye da na yau da kullun, ƙasa da sau da yawa suna haifar da samuwar ƙwayoyin cuta, lipodystrophy, halayen rashin lafiyar jiki.

Hanyar Maganin Insulin

Ana yin lissafin adadin allurai na yau da kullun na insulin cikin la'akari da matakin yawan ƙwayar cutar glucemia da glucosuria. Sauran abubuwa sun zama daidai, ya kamata a kula da kulawa ta musamman lokacin da za a ƙayyade allurai na insulin a cikin yanayin lalacewar koda, tunda ƙananan ƙididdigar glucosuria ba koyaushe suke nuna daidai matakin glycemia ba. Bugu da ƙari, kodan shine wurin lalacewa (lalata) na insulin kuma idan aikinsu ya lalace, buƙatar insulin ya ragu, wanda ke ƙarƙashin gyara na wajibi. In ba haka ba, mai haƙuri, da alama a allurai na insulin a gare shi, na iya haɓaka cutar haɓaka, mai haifar da haɗari ga rayuwa.

Da farko, an sanya wa mara lafiya matsakaicin adadin yau da kullun - wannan ƙimar da ke nuna matsakaicin bukatun yau da kullun don insulin, ya danganta da nauyin jikin mai haƙuri da tsawon lokacin cutar.

Siffofin ciwon sukari na 1

Matsakaicin adadin yau da kullun na insulin, UNITS / kg

Bayan ramawa game da rikice-rikice na rayuwa na nau'in 1 na farko da aka gano

Idan har ba'a biya diyya ba

Na biyu shekara da tsawon tsawon cutar

Ketoacidosis, haɗakar cututtuka da cututtuka masu kumburi

A halin yanzu, suna amfani da hanyar basal-bolus na gudanarwar insulin (i.e., haɗakar insulins-gajere da aiki mai tsawo), suna kwaikwayon ɓarin ilimin insulin na insulin. A wannan yanayin, ana gudanar da aikin insulin na tsawon lokaci kafin karin kumallo a kashi daya daidai da 1/3 na yawan yau da kullun, ana gudanar da ragowar 2/3 na yawan yau da kullun a cikin hanyar insulin gajeren aiki (ana rarraba shi kafin karin kumallo, abincin rana da abincin dare a cikin rabo na 3: 2: 1).

Kulawar gaggawatare da hyperglycemic coma:

Janar na magani ga kamuwa da cutar siga ya haɗa da:

1) kawarda karancin insulin da kuma tsarin metabolism na metabolism,

2) ingantaccen saurin motsa jiki,

3) sabunta kayan al'ada - da kayan haɗin ciki,

4) maido da glucose (glycogen) ajiyar cikin jiki,

5) sabunta daidaitaccen acid-base balance (COR),

6) bincike da lura da cututtuka ko cututtukan cututtukan da suka haifar da cutar sankara,

7) jerin hanyoyin warkewa da nufin dawo da aiki da ayyukan gabobin ciki (zuciya, kodan, huhu, da sauransu).

Don magance rikici a cikin cututtukan masu ciwon sukari, catecholamines da sauran magunguna na juyayi ba za a yi amfani dasu ba. Ba a haɗa Contraindication ba kawai tare da gaskiyar cewa catecholamines sune kwayoyin homonin-contra-hormone, amma kuma tare da gaskiyar cewa a cikin marasa lafiya masu ciwon sukari tasirin tasirinsu a kan ɓoyewar glucagon yana da ƙarfi sosai fiye da lafiyar mutane.

Da zaran an kai mara lafiya zuwa cibiyar likitanci, kafin fara magani, sai su tantance matakin glucose na jini (idan zai yiwu jikin ketone, har da pH, alkaline Reserve, electrolytes da nitrogen reshe), yi aikin ɓoye tare da kafa microcatheter mai ɓarna. Abu na gaba, catheterization na mafitsara da yanke shawara cikin gaggawa a cikin fitsari na matakan glucose da jikin ketone (idan ya yiwu kuma furotin da kwayoyin jini), tilasta lavage na ciki tare da maganin bicarbonate.

Ketoacidotic coma insulin far yana farawa lokaci guda tare da farfadowa, sau da yawa a matakin prehospital. A halin yanzu, a duk ƙasashe na duniya, ciki har da a ƙasarmu, ana amfani da “ƙarancin” insulin na insulin don amfani da waɗannan dalilai. Dalilin yin amfani da "ƙananan" allurai na insulin a cikin ketoacidosis shine binciken da ke nuna cewa matakin insulin jini na 10-20 mU / ml yana hana lipolysis, gluconeogenesis da glycogenolysis, da kuma haɗuwa da 120-180 mU / ml inhibits ketogenesis. Gabatar da insulin a cikin kudi na 5-10 U / h yana haifar da maida hankali a cikin jini, wajibi ne don dakatar da lipolysis, glycogenolysis da glucogenesis, amma har da ketogenesis.

Mafi kyawun ci gaba mai saurin kamuwa da ƙananan allurai na insulin. Ana amfani da insulin mai sauƙi a cikin 0.9% sodium chloride bayani kuma an zuba shi a cikin adadin 5-10 (ƙasa da sau da yawa a cikin 10-15) U / h. Kafin farkon jiko, ana ba da shawarar sassan 10 na insulin cikin kulawa. Matsayin insulin da ake buƙata don ci gaba da jiko sama da awa daya shine 0.05-0.1 U / kg.

Yawan jiko kuma, saboda haka, adadin insulin ya dogara da kuzarin abubuwan da ke cikin glucose a cikin jinin mai haƙuri, wanda ke sa ido a kowace awa. Matsakaicin gwargwadon raguwa a cikin jini shine 3.89-5.55 mmol / h. Bayan glucose na jini ya ragu zuwa 11.1-13.9 mmol / L, an rage yawan kumburin insulin ta hanyar 2-4 U / h wanda wannan alamarin ya kasance cikin kewayon 8.33-11.1 mmol / l don daidaita cikin pH na jini, sannan ana gudanar da insulin a cikin subcutaneously a raka'a 12 a kowane sa'o'i 4 ko kuma raka'a 4-6 kowane 2 hours.

Ana kula da cututtukan ƙwayar cuta, gas da jini, da kuma glucosuria da ketonuria kowace sa'a. Idan har zuwa ƙarshen sa'a na farko na jiko matakin bai rage by 10% daga farkon na farko ba, yana da mahimmanci don maimaita aikin lokaci daya na 10 PIECES na insulin kuma ci gaba cikin jiko a daidai wannan adadin ko ƙara yawan jiko na insulin zuwa 12-15 PIECES / h.

Mayar da glucose a cikin jiki shine mataki na karshe a cikin magance cutar sikari. Kamar yadda aka nuna a sama, tare da raguwa a cikin glycemia zuwa 11.1-13.9 mmol / l, ana rage rage yawan insulin, yayin da aka fara saurin narkewar maganin glucose 5%. A nan gaba, ana yin aikin insulin ne kawai a hade tare da gabatarwar glucose, ta yadda a matakin glycemia sama da mm 10-11 mmol / l, ga kowane 100 na 100 na maganin glucose 5%, ana gudanar da sassan insulin na 2-3, kuma tare da glycemia a ƙasa 10 mmol / l - ba ƙari ba 1 raka'a ta 100 ml na 5% bayani. Ana samar da maganin glucose mai isotonic a cikin adadin 500 ml a cikin sa'o'i 4-6, yayin da adadin glucose da aka gudanar a kowace rana ya kamata ya zama 100-150 g. Tare da saka idanu na dakin gwaje-gwajen da suka dace, wannan tsari na rikitarwa na "glucose insulin" yana ba da damar kula da daidaitaccen ƙwayar glucose jini na 9 -10 mmol / l na dogon lokaci.

Kulawa ta gaggawa don cutar sikila:

Lokacin tabbatar da ganewar asali na maganin hypoglycemic coma, magani ya ƙunshi gabatarwar 50 ml na maganin glucose na 50% a cikin ciki (idan ba zai yiwu ba don maganin abinci na mai haƙuri) na mintina 3-5, biyowa jigilar narkewar 5 ko 10% maganin glucose. A cikin wasu marasa lafiya, dawo da hankali yana faruwa nan da nan bayan gudanarwar glucose, a cikin wasu yana ɗaukar wani lokaci. Gudanar da tasirin glucose yakamata ya ci gaba cikin tsawon lokacin da ake tsammani na yin aiki na insulin ko magani na baki wanda ya haifar da wannan coma (alal misali, idan ana haifar da kwayar cutar ta hanyar shan chlorpropamide, ya kamata a gudanar da glucose tsawon kwanaki). Bugu da ƙari, an bada shawarar gabatarwar 1 mg na glucagon intramuscularly. Bayan dakatar da shan coma, gyaran hanyoyin rage sukari, rage cin abinci da tsarin marasa haƙuri ya kamata a aiwatar.

Menene coma mai ciwon sukari

Ofaya daga cikin rikice-rikice na yau da kullun shine ciwon sukari wanda ke da alaƙa da canji a cikin yawan ƙwayar cutar plasma da haɓakar canje-canje na rayuwa. Idan ba a gano wani a cikin lokaci ba, to canje-canje na iya zama wanda ba a iya juyawa ba har ya kai ga mutuwa.

Hyperglycemic

Hyperglycemic (hyperosmolar) coma a cikin ciwon sukari ana nuna shi da glucose na jini (fiye da 30 mmol / l), sodium mai yawa (fiye da 140 mmol / l), babban osmolarity (yawan narkar da cations, anions da tsaka tsaki abubuwa sun fi 335 sauro / l) .

Me zai iya tsokani:

1. Shan magungunan da ba a sani ba suna rage matakan glucose.
2. Ba da izinin cirewa ko maye gurbin magungunan hypoglycemic, ba tare da shawara tare da likitanka ba.
3. Hanyar da ba daidai ba ta gudanar da kwayoyi masu ɗauke da insulin.
4. Ilimin aikin likita na ciki - rauni, pancreatitis, ciki, tiyata.
5. Mafi mahimmancin carbohydrates a cikin abinci - matakan glucose yana ƙaruwa.
6. Yin amfani da wasu ƙwayoyi (diuretics yana haifar da rashin ruwa, don haka yana ƙaruwar osmolarity, glucocorticoids yana ƙara glucose jini).
7. Rstarma, tare da ɗan adadin ƙwayar da aka cinye. Yana haifar da bushewa.
8. Kwatsam na ruwa, maimaita maimaitawa - rashin ruwa yakan haifar.

Yana cikin haɗari ga rayuwa da lafiya. Tare da saurin ƙaruwa cikin sukari na jini, dole ne a hanzarta neman taimakon likita.

Hyma na jini

Regimen na farko na maganin cutar malaria.

Wannan shine mafi yawan nau'in coma a cikin ciwon sukari. An kwatanta shi da faɗuwa cikin kwatsam cikin jini a ƙasa 3 mmol / L.

• babban allurai na insulin
• rashin nasara
• zafin motsa jiki,
• shan allurai barasa,
• wasu kwayoyi (B-blockers, lithium carbonate, Clofibrate, anabolics, alli).

Sau da yawa yakan faru, amma ana samun sauƙin dakatar da amfani da carbohydrates mai sauri (ruwa tare da sukari, alewa).

Cutar mai fama da cutar sikari

Wannan shine coma mafi hatsari a cikin cututtukan mellitus, wanda pH ya faɗi ƙasa da 7.35, matakan glucose ya karu zuwa 13 ko fiye, kuma adadin adadin jikin ketone yana cikin jini. Mutane masu ciwon sukari na cikin ƙasa sun fi fuskantar wahala. Dalilin shi ne zaɓi na rashin daidaituwa na allurai na insulin ko karuwa a cikin buƙatar hakan.

1. Suarancin maganin cutar yawan kumburi da tsallakewar ƙwayar insulin.
2. Nisantar maganin cututtukan zuciya.
3. Ba daidai ba ne gudanar da shirye-shiryen insulin.
4. Abubuwan da ke tattare da rikice-rikice - ayyukan tiyata, bugun jini, da sauransu
5. Abincin carb, abinci mai tsafta.
6. Aiki mai ƙarfi na jiki tare da bayyane haɗuwa da hawan jini mai yawa.
7. Al'adar fata
8. Wasu magunguna (maganin hana haihuwa, diuretics, morphine, shirye-shiryen lithium, dobutamine, adrenal da thyroid).

Ketoacidotic coma koyaushe yana buƙatar sa hannun likita tare da sake tayar da hankali, in ba haka ba mutum ya mutu.

Bambanci a cikin bayyanar cututtuka

Tebur: Halin kamanceceniya.

Ketoacidotic da hyperglycemic coma na ciwon sukari mellitus suna girma a hankali, mutum zai iya kula da bayyanar ƙanshin wari daga bakin ko raguwar ƙarfin ƙwayar tsoka. Abun cikin jini ya bunkasa sosai, saboda haka ya kamata mai haƙuri koyaushe ya kasance yana da Sweets tare da shi, wanda yakamata a cinye shi lokacin da rawar jiki ya bayyana.

Taimako na farko don maganin cutar rashin motsa jiki na hyperglycemic

Dabaru kafin isowar likitoci:

1. Sanya a gefenta, gyara harshe.
2. Eterayyade idan akwai cutar mellitus na ciwon sukari ko yanayin haɓaka da farko.
3. Idan za ta yiwu, a auna sikirin jini kafin gudanar da aikin insulin da mintina 20 bayan haka. Shigar da raka'a 5-10 na insulin ƙarƙashin ƙasa.
4. Lokacin dakatar da numfashi amfani da hurawa ta wucin gadi ta bakin.
5. Tare da raɗaɗin hana ƙwayoyin jiki.

Ayyuka don rashin lafiyar hypoglycemic

Matakan bayar da agajin farko:

1. Sanya a gefenta, gyara harshe.
2. Yi ƙoƙarin bayar da cikakken sukari mai sha don sha (3 tbsp. Per 100 ml na ruwa) ko saka allurar glucose (magani na kantin magani) a ciki.
3. Lokacin da numfashi ya tsaya, aiwatar da matakan sake tsinkaye - nutsuwa ta wucin gadi ta bakin.
4. Yi ƙoƙarin gano ko mutum yana da ciwon sukari ko kuma idan yanayin ya tashi kwatsam.

Abin da za a yi tare da ketoacidotic coma

1. Sanya mai haƙuri a gefe, gyara harshe.
2. Shigar 5-10 IU na insulin.
3. Lokacin da numfashi ya tsaya, yi numfashi na mutum.
4. Saka idanu yawan zuciya, hawan jini, sukari jini.

Kulawa ta gaggawa tare da ketoacidotic coma ta sauko zuwa jiko farji (sarrafawa na cikin kwayoyi), don haka ya zama likitoci.

Idan ba'a bayyana nau'in coma ba

1. Gano idan mai haƙuri yana da ciwon sukari.
2. Duba matakin glucose.
3. Yi nazarin mutum don bayyanar cututtuka na nau'in coma guda.

Coma yanayi ne mai haɗari, ba zai yiwu a aiwatar da takamaiman matakan warkewa a gida ba. A cikin diabetology, abin da za a yi a cikin irin waɗannan halayen ana bayar da rahoton a cikin algorithms na gaggawa, ga kowane nau'in coma sun bambanta, amma ana iya yin su ne kawai idan an sami ilimin likita.

Taimako na Farko ga Cutar Malaria

Daya daga cikin cututtukan yau da kullun da ke cike da rauni shine ciwon sukari. Da yawa ba su sani ba, saboda rashin bayyana alamun bayyanar cututtuka, cewa suna da ciwon sukari. Karanta: Babban alamun bayyanar cutar sankarau - a wane lokacine za'a kiyaye? Bi da bi, karancin insulin na iya haifar da rikice rikice kuma idan babu ingantaccen magani, ya zama barazanar rayuwa. Mafi yawan rikice-rikice na ciwon sukari coma ne. Wadanne nau'ikan cututtukan cututtukan cututtukan da ke ciki ake sani, da kuma yadda za a iya ba da taimakon farko ga mai haƙuri a wannan yanayin?

Cutar kamuwa da cutar sankarau - babban sanadin hakan, nau'in coma masu cutar sankara

Daga cikin duk rikitar da ciwon sukari, yanayin mai kama da na masu fama da cutar siga shine, a mafi yawancin lokuta, ana iya juyawa. Dangane da mashahurin imani, cutar sankarar mahaifa cuta ce ta amai da gudawa. Wato, wuce haddi na sukari jini. A zahiri, coma mai ciwon sukari na iya zama nau'i daban-daban:

1. Hypoglycemic
2. Hyperosmolar ko ƙwaƙwalwar ƙwayar cuta
3. Ketoacidotic

Sanadin coma mai ciwon sukari na iya zama karuwa mai yawa a yawan glucose a cikin jini, kulawa mara kyau ga masu ciwon sukari har ma da yawan insulin, wanda yawan sukari ya ragu a kasa.

Bayyanar cututtuka na cutar hypoglycemic coma, taimako na farko don cutar rashin ƙarfi na hypoglycemic

Yanayin hypoglycemic halayyar ne, don mafi yawan bangare, don nau'in ciwon sukari na 1, kodayake suna faruwa ne a cikin marasa lafiya waɗanda ke shan kwayoyi a allunan. A matsayinka na mai mulkin, ci gaban jihar ya gabata hauhawar yawan insulin a cikin jini. Hadarin hypoglycemic coma yana cikin nasara (ba a sake juyawa) na tsarin juyayi da kwakwalwa.

Taimako na farko don maganin cutar rashin ruwa na hypoglycemic

Tare da m alamu ya kamata mai haƙuri ya gaggauta ba da piecesan guda na sukari, kusan 100 g na kukis ko 2-3 tablespoons na jam (zuma). Yana da kyau a tuna cewa yayin da ciwon sukari mai dogaro da insulin yakamata a koyaushe a sami wasu abubuwan lemun zaƙi “a ƙirjin”.
Tare da alamu mai tsanani:

• Zuba shayi mai ɗumi a cikin bakin mai haƙuri (gilashin / cokali 3 na sukari) idan zai iya hadiye shi.
• Kafin jiko na shayi, yana da mahimmanci don saka mai riƙewa tsakanin hakora - wannan zai taimaka don kauce wa matsi mai ƙarfi.
• Dangane da haka, darajar ingantawa, ciyar da mai haƙuri abincin mai arziki a cikin carbohydrates ('ya'yan itãcen marmari, kayan abinci da hatsi).
• Don guje wa hari na biyu, rage kashi na insulin ta hanyar raka'a 4-8 a safiyar gobe.
• Bayan kawar da tsotsar jinin haila, nemi likita.

Idan coma tasowa tare da asarar sanisannan kuma ya biyo baya:

• Gabatar da 40-80 ml na glucose a cikin ciki.
• Da sauri kira motar asibiti.

Taimako na farko don maganin cutar hyperosmolar

• Daidai sa haƙuri.
• Gabatar da karkatar da magana da kuma hana fitar da harshe.
• Yi gyare-gyare na matsin lamba.
• Gabatar da ciki na 10-20 ml na glucose (maganin 40%).
• A cikin maye sosai - a kira motar asibiti nan da nan.

Kulawa ta gaggawa don cutar ketoacidotic, alamomin da ke haifar da cutar ketoacidotic a cikin ciwon sukari

Dalilaiwanda ke kara buƙatar insulin kuma suna ba da gudummawa ga haɓaka ƙwayar ketoacidotic yawanci:

• Late ciwon sukari da ciwon sukari.
• Wanda ba shi da magani da aka wajabta (ya sanya magani, sauyawa, da sauransu).
• Jahilcin ka'idodin sarrafa kai (yawan shan barasa, raunin abinci da ƙa'idodin aiki na jiki, da sauransu).
• Cutar cututtukan mahaifa.
• Raunin jiki / kwakwalwa.
• Cutar cututtukan jijiyoyin jiki a cikin siffar m.
• Ayyuka.
• Rashin haihuwa / haihuwa.
• Damuwa.

Cutar Ketoacidotic - alamomi

Alamar farko zama:

• Urination akai-akai.
• Tsammani, tashin zuciya.
• Damuwa, rauni gaba ɗaya.

Tare da bayyana tabarbarewa:

• Sell ​​na acetone daga bakin.
• M zafi ciki.
• Matsanancin amai.
• Rashin ƙarfi, numfashi mai zurfi.
• Sa'annan kuma yana shigowa, yana da illa, zai iya faduwa zuwa yanayin rayuwa.

Cutar Ketoacidotic - taimakon farko

Da farko dai Ya kamata a kira motar asibiti ta duba duk mahimman ayyukan mai haƙuri - numfashi, matsin lamba, bugun zuciya, hankali. Babban aikin shine tallafawa bugun zuciya da numfashi har sai motar asibiti ta isa.
Gane ko mutum ya waye, zaka iya ta hanya mai sauki: ka tambaye shi kowace tambaya, dan kadan ya buge a kan kunci da shafa kunnuwa na kunnuwansa. Idan babu dauki, mutumin yana cikin haɗari babba. Saboda haka, jinkirta kiran motar motar asibiti bashi yiwuwa.

Gabaɗaya ƙa'idodi don taimakon farko na cutar masu ciwon sukari, idan ba a bayyana nau'in sa ba

Abu na farko da dangin mai haƙuri yakamata suyi tare da asali kuma, musamman, alamun alamun rashin damuwa shine kira motar asibiti nan da nan . Marasa lafiya masu ciwon sukari da danginsu sun saba da wadannan alamu. Idan babu yiwuwar zuwa likita, to a farkon alamun yakamata ku:

• Intramuscularly allurar insulin - raka'a 6-12. (ba na tilas ba ne).
• Doseara kashi gobe da safe - raka'a 4-12 / a lokaci guda, inje 2-3 a rana.
• Ya kamata a kwarara yawan abincin Carbohydrate, fats - ware.
• Theara yawan 'ya'yan itatuwa / kayan lambu.
• Amfani da ruwan ma'adinan alkaline. A cikin rashi - ruwa tare da narkar da cokali na shan soda.
• Enema tare da maganin soda - tare da rikicewar hankali.

'Yan uwan ​​mai haƙuri yakamata suyi nazarin halaye na cutar, magani na zamani da ciwon sukari, diabetology da taimakon farko na lokacin - kawai taimakon gaggawa na yau da kullun zai yi tasiri.