2) Guban jini

Glycemia - matakin glucose a cikin jini. Ka'ida shine 60-100 mg% ko 3.3-5.5 mmol / L.

Glycemia ana sarrafa shi ta hanyar hanyoyin da yawa. Matakan glucose na gudana zuwa mafi girma bayan fitarwa, saboda yawan ciki da na hanji na ƙwayoyin carbohydrates mai narkewa mai sauƙi (ƙarancin ƙwayar ƙwayar ƙwayar jiki) daga abinci ko ta hanyar gushewa daga wasu abinci kamar taurari (polysaccharides). Matsayin glucose yana raguwa sakamakon catabolism, musamman tare da kara yawan zafin jiki, tare da ƙoƙarin jiki, damuwa.

Sauran hanyoyi don tsara glycemia sune gluconeogenesis da glycogenolysis. Gluconeogenesis shine tsarin samar da kwayoyin glucose a cikin hanta kuma wani bangare a cikin abubuwan cortical na kodan daga kwayoyin sauran kwayoyin, misali, amino acid, lactic acid, glycerol. Yayin glycogenolysis, glycogen da ya tara na hanta da kasusuwa kwarangwal an canza shi zuwa glucose ta hanyar sarƙoƙi da yawa.

Ana canza glucose mai wuce haddi zuwa glycogen ko triglycerides don adana makamashi. Glucose shine tushen samar da makamashi na yau da kullun ga yawancin sel, musamman ga wasu sel (alal misali, neurons da farin jini), wadanda kusan sun dogara da matakan glucose. Kwakwalwar tana buƙatar glycemia mai natsuwa don ya yi aiki. Haɗuwar glucose na jini ƙasa da 3 mmol / L ko fiye da 30 mmol / L na iya haifar da rashin sani, tashin hankali, da kuma coma.

Abubuwa da yawa suna shiga cikin tsarin glucose na rayuwa, kamar insulin, glucagon (wanda ke dauke da hancin), adrenaline (glandon adrenal ta dauke shi), glucocorticoids da kwayoyin hodar iblis (wadanda ke gonads da na hanji adrenal).

m hyperglycemia - 6.7-8.2 mmol / l,

matsakaici mai ƙarfi - 8.3-11.0 mmol / l,

nauyi - fiye da 11.1 mmol / l,

tare da nuna alama na 16.5 mm / l, precoma yana haɓaka,

tare da mai nuna alama akan 55.5, ƙwayar hyperosmolar na faruwa.

Babban dalilin faruwar cutar hyperglycemia shine karancin insulin (wani kwayar halittar dake rage yawan motsa jiki a cikin jini). Wasu lokuta, insulin kuma bazai iya hulɗa da jikin mutum daidai ba don amfani da glucose.

Akwai dalilai da yawa don haɓakar haɓakar hyperglycemia, daga cikinsu suna yawan wuce gona da iri, cin abinci mai-kalori mai yawa wanda ya ƙunshi adadin karfan carbohydrates mai sauƙi da rikitarwa.

Damuwar damuwa na iya zama sanadin rashin ciwon sukari wanda ke haifar da hauhawar jini. Wajibi ne don sarrafa aikinku: aiki mai yawa ko, akasin haka, salon rayuwa yana iya haifar da hauhawar sukarin jini.

Cutar cututtukan daji da na kullum na iya haifar da bayyanar cututtukan hyperglycemia. A cikin mutanen da ke fama da ciwon sukari, hyperglycemia na iya faruwa saboda magunguna masu rage sukari da aka rasa ko allurar insulin.

- karancin jini.

2) mara kyau abinci mai gina jiki tare da zagi na carbohydrates mai ladabi, tare da raunin ƙwayar firam, bitamin, salts ma'adinai,

3) lura da ciwon sukari mellitus tare da insulin, maganganu na hypoglycemic na maganganu idan aka samu yawan yawan haɗuwa,

4) karancin abinci ko makara,

5) yawan motsa jiki,

7) haila a cikin mata,

9) mummunan gajiyawar gabobin jiki: renal, hepatic ko ciwon zuciya, sepsis, ci,

10) karancin matsalar ciki: cortisol, hormone girma, ko duka biyun, glucagon + adrenaline,

ba tarin kwaya-p-cell ba,

11) ciwace (insulinoma) ko rashin lafiyar haihuwar yara - 5-cell hypersecretion, autoimmune hypoglycemia, 7-ectopic insulin ɓoye,

12) hauhawar jini a jarirai da yara,

13) sarrafa ruwan ciki mai narkewa tare da daskararre.

Wasarshen gyaran da aka yiwa wannan shafin shine: 2017-01-24, Shafin Infetare Hakkin Mallaka

1) Glycolysis. Matsayin halittu, ilmin sunadarai na tsari, nazarin halittu, tsari. Tasirin Pasteur.

shine barkewar kwayar cutar anaerobic zuwa ga lactate.

C6H12O6 + 2ADP + 2Fn = 2 lactate + 2ATP + 2H20.

ya hada da halayen 11 da matakai 2.

Saboda glycolysis, jiki yana yin ayyuka da yawa a cikin yanayin ƙarancin oxygen.

Lokacin da babu oxygen a duniya, glycolysis shine asalin tushen ƙarfin.

Enzymes Glycolysis suna cikin gida a cikin cytoplasm.

- Mafi tsananin zafin nama a cikin:

-3 halayen da ba za'a iya juyawa ba (kinase).

Mataki na farko na glycolysis

Mataki na biyu na glycolysis

Cibiyar mai aiki da enzyme glyceraldehyde foshate dehydrogenase ta ƙunshi SH-rukuni na cysteine.

A matakin farko, an cire hydrogen daga rukunin aldehyde na substrate, na biyu kuma shine hydrogen daga kungiyar SH-rukunin cibiyar aiki.

Hydrogen yana wucewa zuwa NAD, a sakamakon muna samun NADH + H +, an samar da wani hadadden enzyme-substrate, wanda ke hulda da acid din.

Energyarfin kuzari da aka saki yayin iskar shaka na ƙungiyar aldehyde an adana shi a cikin rukunin phosphate mai ƙarfi.

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